Patient demonstrated increase perfusion with warm skin, absence of edema, free of pain

A complication of any form of shock caused by inadequate tissue perfusion. The precise mechanism by which MODS occurs remains unknown. Occurs toward the end of the continuum of septic shock when tissue perfusion cannot be effectively restored. It is not possible to predict which patients who experience shock will develop MODS, partly because much of the organ damage occurs at the cellular level and therefore cannot be directly observed or measured. However, a pattern of progressive organ dysfunction and failure typically occurs; organ failure usually begins in the lungs, and cardiovascular instability as well as failure of the hepatic, GI, renal, immunologic, and central nervous systems follow. Advanced age, malnutrition , and coexisting disease appear to increase the risk of MODS in acutely ill patients.

HISTORY. The patient with MOF has a history of infection, tissue injury, or a perfusion deficit to an organ or body part.

Administer oxygen as prescribed Restrict patients fluid intake Instruct the patient to eat low fat low sodium diet and high in carbohydrates.

MULTIPLE ORGAN FAILURE

Question the patient (or, if the patient is too ill, the family) Elicit complete medication history and patients compliance Ask if the patient has experience recent weight loss Determine the patients dietary patterns. Take a hx of patients use of cigarettes, alcohol, and other drug abuses

Primary nursing interventions are aimed at supporting the patient and monitoring organ perfusion until primary organ insults and halted Patient will increase perfusion as evidence by warm and dry skin, peripheral pulses present and strong absence of edema, free of pain and discomfort.

Ineffective tissue perfusion related to decrease cellular oxygenation

Clinical signs and symptoms: Cardiovascular: hypotension and hypoperfusion Respiratory: hypoxemia, hypercarbia, adventitious breath sounds Renal: increased creatinine, decreased UO Hematologic: thrombocytopenia, coagulation abnormalities Metabolic: lactic academia, metabolic acidosis Neurologic: altered LOC Hepatic: elevated liver function tests, hyperbilirubinemia