Professional Documents
Culture Documents
a. Liver
b. Salivary glands
c. Spleen
d. Heart
e. Bladder
a. AST
b. ALT
c. Alkaline phosphatase
d. 5′-Nucleotidase
e. γ-Glutamyl transpeptidase
a. Blood transfusion
b. Intravenous drug abuse
c. Fecal-oral route
d. Sexual
a. HAV
b. HBV
c. HCV
d. HEV
e. HGV
a. Anti-HBe
b. Anti-HBc
c. Anti-HBs
d. Anti-polymerase
e. Anti-HBV DNA
9.A 51-year-old man health care worker whom you examine for the
first time feels well. You do a complete physical examination,
which is normal except for slight overweight and borderline
hypertension. It is interesting that his laboratory studies show the
following hepatitis B virus profile: positive HBsAg, negative anti-
HBs, low levels of IgG anti-HBc, positive anti-HBeAg, and negative
anti-HBe. The likely diagnosis is
a. Acute HBV infection, high infectivity
b. Late-acute HBV, low infectivity
c. Recovered from HBV infection
d. Chronic HBV infection, high infectivity
e. Immunization with HBsAg vaccine
a. A
b. B
c. C
d. E
e. G
a. Hemochromatosis
b. Gaucher’ s disease
c. Biliary cirrhosis
d. Wilson’ s disease
e. Type III glycogen storage disease
a. Hemochromatosis
b. Gaucher’ s disease
c. Biliary cirrhosis
d. Wilson’ s disease
e. Type III glycogen storage disease
a. HAV
b. HBV
c. HCV
d. HEV
e. HGV
a. Acute hepatitis A
b. Acute hepatitis B
c. Acute hepatitis A and B
d. Chronic hepatitis B
e. Acute hepatitis C
a. Acute hepatitis A
b. Acute hepatitis B
c. Acute hepatitis A and B
d. Chronic hepatitis B
e. Acute hepatitis C
a. Constipation
b. Hyperkalemia
c. Gastrointestinal bleeding
d. Hypernatremia
e. Acidosis
a. Tuberculosis
b. Peritoneal metastases
c. Cirrhosis of the liver
d. Trauma
a. Neonatal jaundice
b. Crigler-Najjar syndrome, types I and II
c. Hepatitis
d. Partial extrahepatic obstruction
e. Intravascular and extravascular hemolysis
a. Neonatal jaundice
b. Crigler-Najjar syndrome, types I and II
c. Hepatitis
d. Gilbert’ s syndrome
e. Ineffective erythropoiesis
a. Sucrose
b. Lactulose
c. Protein
d. Glucose
e. Galactose
29.Which one of these drugs is of proven benefit in the treatment
of persons with hepatic encephalopathy
a. Levodopa
b. Bromocriptine
c. Keto analogues of essential amino acids
d. Branched-chain amino acids
e. Tetracycline
a. HAV
b. HBV
c. HDV
d. HEV
e. HGV
33.Which disease is a major risk factor for development of
hepatocellular carcinoma
a. Alcoholism
b. Coombs’ positive hemolytic anemia
c. Cholelithiasis
d. Cirrhosis
e. Amyloidosis
a. Albumin
b. α-Fetoprotein
c. Thyroxine-binding globulin
d. Protein C
e. C-reactive protein
a. Cirrhosis
b. Wilson’ s disease
c. Hepatitis due to HAV
d. Hepatitis due to HBV
e. Hepatitis due to HCV
a. Cirrhosis
b. Wilson’ s disease
c. Hepatitis due to HAV
d. Hepatitis due to HBV
e. Hepatitis due to HCV
a. HAV
b. HBV
c. HCV
d. HDV
e. HGV
39.Hepatocellular adenomas
a. Hemochromatosis
b. Gaucher’ s disease
c. Biliary cirrhosis
d. Wilson’ s disease
e. Type III glycogen storage disease
1-a , 2-c , 3-c , 4-b , 5-d , 6-c , 7-b , 8-c , 9-d , 10-c , 11-b , 12-b ,
13-d , 14-d , 15-a , 16-a , 17-e , 18-c , 19-c , 20-b , 21-c , 22-e , 23-d
24-c , 25-c , 26-e , 27-c , 28-b , 29-e , 30-d , 31-c , 32-b , 33-d ,
34-b , 35-c , 36-e , 37-a , 38-a , 39-a , 40-a
a) Hepatitis A virus
b) Prolonged alcohol consumption
c) Gastric bypass surgery
d) Type 2 diabetes
e) Long lasting total parentral nutrition
a) Jaundice
b) Ascites
c) Enlarged tender liver
d) Positive hepatojugular reflux
e) Liver failure
answers : 1-e , 2-d , 3-b , 4-b , 5-c , 6-b , 7-c , 8-d , 9-c , 10-e , 11-c , 12-a , 13-d , 14-d.