By Dr. Ali H. Sadiek Prof. of Internal Veterinary Medicine and Clinical Laboratory Diagnosis
Dept. of Animal Medicine
Faculty of Veterinary Medicine Assiut University- Assiut, EGYPT
E-mail: Sadiek59@yahoo.com
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Diseases of the Oral cavity .Lip & Gum Exam• Swelled cheek•
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I- Stomatitis Inflammation of mouth cavity, tongue, gums, palates. It is cH. by: • Anorexia, Excessive salivation • Mouth lesions (Abrasions, vesicles, pustules, ulcers etc.) • Swellings of adjacent L. Nodes. • It may be primary or sec. to other diseases ( Indigesion, vit. C def.) or assoc. FMD, RP, POX MHCV, VS, etc).
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Causes and types of Stomatitis 1-Bacterial stomatitis oral necrobacillosis: usually necrotic and is manifested by ulceration and suppuration. caused by spherophorus necrophorus. 2-Mycotic stomatitis: infection with Monilia spp. fungi. It usually form a heavy white deposit with little inflammation or damage to the mucosa.
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3- Viral stomatitis: b) Vesicular stomatitis: Vesicular lesions are thin-walled vesicles 1-2 cm in diameter, filled with clear serous fluid. When rupture it leaves sharp edged shallow ulcers as in cases of FMD, VSt. and V.Exanth. d) Erosive stomatitis: Erosive lesions are shallow, usually discrete areas of necrosis. lesions occurs commonly on the lingual mucosa and at the commissars of the mouth. The lesions penetrate deeply to the lamina propria. Prof. Dr. Ali H. Sadiek 5 3- Viral stomatitis: The erosive and secondary ulcerative stomatitis occurs in: Rinderpest, mucosal disease, blue tongue, infectious ulcerative stomatitis. Ulcerative dermatitis (ORF), sheep-pox and contagious exanthema are primarily skin diseases but may involve the alimentary tract including the oral cavity. Proliferative form occur in proliferative stomatitis, papular stomatitis and in rare cases of papillomatosis.
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Clinical signs of Stomatitis 1- Excessive salivation,
Treatments of stomatitis In primary stomatitis: Antispetics: 2 % Copper sulphates, 2 % Borax, 1 % Sulpha and glycerin, 1 % Acriflavin and glycerin. In Ulcerative stomatitis: Curate ulcers and apply silver nitrate. In all cases I.V./I.M. antibiotics if necessary. Separate feeding with easily digestible food or parentrally fed. :In secondary stomatitis Managed according to each diseased condition Prof. Dr. Ali H. Sadiek 12 (II- Glossoplegia (Tongue paralysis ِAll Inflammatory diseases and trauma may also result in transient glossoplegia. species
Newborns As a result of the placement of obstetric snares
In cattle May assoc. severe actinobacillosis
Complete tongue paralysis and necrosis of the tip may be seen in outbreaks in feedlot cattle and may follow viral stomatitis. In Horse It may follow strangles, upper respiratory infections
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II- Glossoplegia (Tongue paralysis) • Central Glossoplegia may follow meningitis, botulism, encephalo-myelitis, leuko- encephalomalacia, or cerebral abscessation in horses. Management and treatment: • Prepheral or central glossoplegia should be managed carefully to ensure eating and ingestion of colostrum. • IV Fluid therapy and anti-inflammatory. • Prognosis is unfavorable, If it persists for >10 days after birth. Prof. Dr. Ali H. Sadiek 14 II- Glossoplegia (Tongue paralysis)
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III- Lip, Mandible and tooth affections
• Most common in horses.
• They may be caused by a fall, a kick, or any trauma. • It may be accompanied by mandibular or incisive bone and dental fractures • Healing is usually rapid. However, once penetrated fistula may occur. Prof. Dr. Ali H. Sadiek 16 -IVSlaframine Toxicosis • Caused by ingestion of forages, e.g clovers, infected with the fungus Rhizoctonia leguminicola , which produces the toxic alkaloid slaframine. • Diagnostic signs are profuse salivation; no mouth lesions. • Differential diagnoses bluetongue, ves. stomatitis, ves.exanthema, and FMD. • Removal of infected forages results in rapid recovery. Prof. Dr. Ali H. Sadiek 17