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BHARATHREDDY
THE CONCEPT OF FASCIAL SPACES IS BASED ON ANATOMISTS KNOWLEDGE THAT ALL SPACES EXIST POTENTIALLY, UNTIL FASCIAE ARE SEPARATED BY PUS, BLOOD, DRAINS OR SURGEONS FINGER.
WHEN DENTAL INFECTIONS SPREAD DEEPLY INTO SOFT TISSUE RATHER THAN EXITING THROUGH ORAL OR CUTANEOUS ROUTES,FASCIAL SPACES MAY BECOME INVOLVED FOLLOWING PATH OF LEAST RESISTANCE.
ON FACE:
SUPRAHYOID:
SUBLINGUAL SUBMANDIBULAR SUBMAXILLARY SUBMENTAL PHARYNGOMAXILLARY PERITONSILLAR
INFRAHYOID:
ANTEROVISCERAL (PRETACHEAL)
FASCIAL LAYER
SUPERFICIAL LAYER
DEEP LAYER
THESE DEVIDE, UNITE, BLEND AND FUSE TO FORM VARIOUS COMPARTMENTS OR SPACES
BUCCAL SPACE
BOUNDRIES
SUPERIORLY INFERIORLY ANTERIORLY POSTERIORLY MEDIALLY LATERALLY ZYGOMATIC ARCH LOWER BORDER OF MANDIBLE MODIOLUS OF MOUTH PTERYGOMANDIBULAR RAPHE BUCCINATOR MUSCLE AND BUCCOPHARYNGEAL FASCIA : SKIN OF CHEEK : : : : :
CONTENTS
SOURCE OF INFECTION
MAXILLARY AND MANDIBULAR MOLAR REGION OR EVEN BICUSPID
IF CONFINED TO BUCCINATOR: INFECTION DRAINS INTRA ORALLY IN BUCCAL VESTIBULE CROSSES BUCCINATOR: INFECTION DRAIN DEEP INTO BUCCAL SPACE AND EXTRA ORAL DRAINAGE
CANINE SPACE
INFREQUENTLY INVOLVED IN ODONTOGENIC INFECTIONS
LEVATOR ANGULI ORIS OVERLIES THE APEX OF CUSPID ROOT . ORIGIN OF THE MUSCLE IS HIGH IN CANINE FOSSA WHEREAS ITS INSERTION IS THE ANGLE OF MOUTH AND ZYGOMATIC MUSCLE. IF CUSPID INFECTION PERFORATES THE LATERAL CORTEX OF MAXILLARY BONE SUPERIOR TO INSERTION OF MUSCLE POTENTIAL CANINE SPACE BECOME INVOLVED
MASTICATOR SPACES
CONSIST OF MESSETERIC PTERYGOID TEMPORAL THESE ARE WELL DIFFERENTIATED BUT COMMUNICATE WITH EACH OTHER AND WITH BUCCAL, SUBMANDIBULAR AND PARAPHARYNGEAL SPACES
SORCE OF INFECTION
THIRD MOLAR (PERICORONITIS, DENTAL CARIES INDUCED ABSCESS ETC)
INFECTION OF MAXILLARY CANINE USUALLY PRESENT AS LABIAL SULCUS SWELLING AND LESS COMMONLY AS PALATAL SWELLING
ALSO BY CONTAMINATED MANDIBULAR BLOCK INJECTIONS OR DIRECT TRAUMA HERE, CLINICALLY THE HALLMARK OF INFECTION IS TRISMUS
SUBLINGUAL SPACE
BILATERAL V SHAPED SPACE
BOUNDRIES:
SUPERIORLY : SUBLINGUAL MUCOUS MEMBRANE INFERIORLY : MYLOHYOID MUSCLE POSTERIORLY : HYOID BONE ANTERIORLY : LINGUAL SURFACE OF MANDIBLE LATERALLY : LINGUAL SURFACE OF MANDIBLE MEDIALLY : GENIOGLOSSUS, GENIOHYOID,
COMMUNICATIONS
ANTERIORLY SPACE : SUBMENTAL
SOURCE OF INFECTION
PREMOLARS PERIODONTAL INFECTION OF INCISORS LINGUAL INJECTIONS INFECTION OF WHARTSONS DUCT SIALIDINITIS
NO EXTRA ORAL DRAINAGE, ONLY INTRA ORAL DRAINAGE D/D: CELLULITIS WITH INFECTED SIALOLITH
SUBMANDIBULAR SPACE
SEPARATED FROM OVERLYING SUBLINGUAL SPACE BY MYLOHYOID MUSCLE
BOUNDRIES
LATERALLY : SUBMANDIBULAR SKIN,SUPERFICIAL FASCIA,PLATYSMA, LOWER BORDER OF MANDIBLE : MYLOHYOID, HYPOGLOSSUS. STYLOGLOSSUS INFERIORLY OF : ANTERIOR AND POSTERIOR BELLY DIGASTRIC
MEDIALLY
CONTENTS
SUBMANDIBULAR SALIVARY GLAND AND LYMPH NODES FACIAL ARTERY WHARTSONS DUCT
LINGUAL NERVE
HYPOGLOSSAL NERVE
SOURCE OF INFECTION
MANDIBULAR SECOND AND THIRD MOLAR SOMETIMES EVEN FIRST MOLAR SECONDARY TO ADJOINING SPACESSUBLINGUAL OR SUBMENTAL
D/D: ACUTE SIALADENITIS SUBMANDIBULAR LYMPHADENITIS
SUBMENTAL SPACE
BOUNDRIES
SUPERIORLY BORDER OF INFERIORLY MUSCLE : INFERIOR MANDIBLE : MYLOHYOID
SOURCE OF INFECTION
MANDIBULAR INCISORS OR FROM SUBMANDIBULAR SPACE
Presentation
The patient presents with a swollen face and occasionally swollen neck. Toothache or facial pain may or may not be a feature. There is often general malaise and possibly rigors with fever. Patients may complain of trismus (inability to open the mouth fully), pain or difficulty in swallowing, drooling, sore throat and a hoarse voice.
Examination
Specific attention should be paid to the location of swelling, size, flactuance, any possible pointing and coexistent lymph node enlargement.
Suspect teeth should be tapped with a metallic object to elicit any tenderness to percussion. Swelling should be palpated bimanually if possible with a finger of one hand intraorally and and the second hand extraorally (pushing towards the oral site). The neck should be evaluated for swelling, lymphadenopathy and possible tracheal deviation.
(1) Decay (caries) reaching the dental pulp=pulpitis, this in turn spreads to supporting bone resulting in (2) periapical abscess which in turn may spread subperiosteally. (2) Periapical abscess may occur in seemingly intact but devitalised teeth (trauma, cracks or decay under fillings). (3) Periapical and periodontal abscess may form as a result of chronic gingivitis and supporting bone and soft tissue loss (periodontal disease) - note again the tooth may be entirely intact clinically and radiographically.
(4) Erupting teeth (especially partially impacted lower third molars) can result in inflammation and infection of the gum flap preventing eruption (operculum) with swelling pus etc. around the crown (pericoronitis). (5) Retained roots supragingival or subgingival.
JAWS
(1)Can develop cysts or tumours that can range from odontogenic (=dental origin) to either primary or secondary malignancy. Most are derived from the dental apparatus and although benign can nevertheless continuously grow and become secondarily infected on breaching the surrounding bone. (2)Osteomyelitis although rare can be the result of chronic infection as mentioned before. (3) Osteoradionecrosis occurs readily in irradiated jaws subjected to further trauma (such as extractions).
(4) Rarer are tuberculosis, Actinomycosis and syphilitic osteomyelitis. (5) Most jaw fractures in the tooth bearing segments are by definition compound to the oral cavity and can easily be infected by the oral microbes. (6) Extraction sites again are comparable to compound fractures and it is surprising that infection is so relatively rare.
Paranasal sinuses
(1)May be primarily infected, obstruct and result in facial swelling. (2)May become infected secondary to infected teeth protruding into the maxillary sinus (upper premolar and molar teeth often do). (3)Tumours or cysts may become infected. (4)Fractures such as the orbital floor are by definition compound to the outside and may result in orbital
Investigations
In many cases careful history and examination will make diagnosis clear, however certain investigation will still be necessary.
Plain X rays
(1)The OPG (orthopantomogram) is invaluable in displaying the teeth, whole of mandible, tooth bearing segment of the maxilla as well as parts of the maxillary sinuses. Use for any suspected fractures of the mandible, periapical abscesses and bony cysts and tumours. Will show impacted third molars ('wisdom teeth'). (2)Occipito-mental 15 and 30 degrees (Waters view) will show both maxillary sinuses (effusion?), orbital floor and most fractures of the maxilla.
(3) Mandibular occlusal views and lateral oblique views may demonstrate stones in the submandibular gland. (4) 'Puffed cheek' view may demonstrate stones in the parotid duct.
Sialography:
Can be used for suspected gland obstruction however CT sialogram is the gold standard.
Ultrasound
Useful in confirming collections as well as a guide to aspiration. Will also show stones in salivary ducts and glands.
CT scan
With axial and coronal views will demonstrate exact extent of the swelling, potential airway compromise and is invaluable to both the surgeon and anaesthetist. However patients unwell enough to potentially obstruct their airway should be taken straight to theatre rather than risk an emergency in the radiology dept.
BUCCALY MESSETRIC SPACE POTENTIAL SPREAD OF INFECTION FROM LOWER THIRD MOLAR
PTERYGOMANDIBULAR SPACE
LATERAL PHARYNGEAL SPACE RETROPHARYNGEAL SPACE MEDIASTINUM CAROTID SHEATH DANGER SPACE 4
NOTE : DANGER SPACE 4 IS THE SPACE BETWEEN PREVERTIBRAL AND ALAR FASCIA
AN INITIAL PERIOD OF PERIAPICAL CONTAMINATION BY BACTERIA GENERALLY ORIGINATING FROM ROOT CANAL CLINICAL PERIOD WITH SIGNS AND SYMPTOMS ACUTE APICAL PERIODONTITIS, DEVELOMENT OF A PERIAPICAL ABSCESS PERIOSTEUM RUPTURES AND INFECTION GAINS ECCESSTO SURROUNDING SOFT TISSUES PRODUCING CELLULITIS ( PHLEGMON ) FINAL RESOLUTION PERIOD AND GENERATION OF REPAIR TISSUE.
CELLULITIS(PHLEGMON)
TYPES 1. SEROUS CIRCUMSCRIBED ACUTE CELLULITIS AFFECTING SINGLE
ANATOMIC SPACE
4. CHRONIC CELLULITIS
CLINICAL MANIFESTATIONS
SHARP PULSATILE PAIN REDENING AND WARMTH OF SKIN AND MUCOSA POORLY DELIMITED SWELLING THAT ERASES THE SKIN FOLDS AND SULCI LOSS OF FUNCTION FEVER
LUDWIGS ANGINA
FIRST DESCRIPTION IN 1836 BY DR.VON LUDWIG ANGINA: CHOAKING SENSATION DEFINITION ARCHER: ITS A BILATERAL,ACUTE,RAPIDLY SPREADING, SEPTIC,INFLAMMATORY,INDURATED ,WOODEN HARD CELLULITIS OF FLOOR OF MOUTH
THOMA: ITS A GANGRENOUS CELLULITIS OF LOOSE ALVEOLAR TISSUE WHICH ORIGINATES IN SUBMANDIBULAR SPACE AND SPREADS RAPIDLY TOWARDS FLOOR OF MOUTH KILLEY-KEY-SEWARD: ITS A CLINICAL DIAGNOSIS AND IS THE NAME GIVEN TO BRAWNY CELLULITIS OCCURING BILATERALLY AT SUBMANDIBULAR REGION WHICH ALSO INVOLVE SUBLINGUAL SPACE
SPREAD OF INFECTION
ACCORDING TO KRUGER,TOPAZIAN,LUDWIG THIRD MOLARS SUBMANDIBULAR SPACE SUBLINGUAL
CONTRALATERAL SUBMANDIBULAR AND SUBMENTAL SPACE INVOLVEMENT LASKIN SUBLINGUAL SPACE SPREADS BILATERALLY SUBMANDIBULAR AND SUBMENTAL SPACE
BACKWARD SPREAD TO SUBSTANCE OF TONGUE INFECTION REACHES EPIGLOTTIS SWELLING AROUND LARYNGEAL INLET MICROORGANISM INVOLVED ARE MOJORITILY STREPTOCOCCUS HEMOLYTICUS
ETIOLOGY PERODONTAL, PERICORONAL OR PERIAPICAL ABSCESS OF MANDIBULAR MOLARS NON ODONTOGENIC CAUSES (PSEUDO LUDWIG) COMPOUND FRACTURE OF MANDIBLE NEEDLE INJURY TO FLOOR OF MOUTH FISH BONE INJURY SIALIDINITIS
LUDWIGS ANGINA
SEQUELE
IT CAN CAUSE MEDIASTINITIS LEADING TO ASPIRATION PNEUMONIA AND DEATH DUE TO RESPIRATORY PARALYSIS IT CAN INVOLVE PTERYGOID COMPARTMENT AND VIA PTERYGOID PLEXUS CAN CAUSE CAVERNUS SINUS THROMBOSIS IT CAN CAUSE SEPTICEMIA OR BACTEREMIA BECAUSE OF HEMATOLOGICAL SPREAD
GENERAL MANAGEMENT
PROPER HISTORY TAKING AND EXAMINATION AND INVESTIGATIONS ANTIBIOTIC ANALGESIC THERAPY ANTIINFLAMMATORY DRUGS FLUID BALANCE AND AIRWAY ESTABLISHMENT WHERE REQUIRED REMOVAL OF FOCUS OF INFECTION ESTABLISHMENT OF DRAINAGE ADEQUATE MEDICAL CONSULTATION AND REFFERAL
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