Professional Documents
Culture Documents
INTRODUCTION
Upper respiratory tract infection (URI) is a nonspecific term used to describe
acute infections involving the nose, paranasal sinuses, pharynx, larynx, trachea, and
bronchi. The prototype is the illness known as the common cold, which will be
discussed here, in addition to pharyngitis, sinusitis, and tracheobronchitis. Influenza is a
systemic illness that involves the upper respiratory tract and should be differentiated
from other URIs.
Viruses cause most URIs, with rhinovirus, parainfluenza virus, coronavirus,
adenovirus, respiratory syncytial virus, coxsackievirus, and influenza virus accounting
for most cases. Human metapneumovirus is a newly discovered agent causing URIs.
Group A beta-hemolytic streptococci (GABHS) cause 5% to 10% of cases of pharyngitis
in adults. Other less common causes of bacterial pharyngitis include group C betahemolytic
streptococci,
Corynebacterium
diphtheriae,
Neisseria
gonorrhoeae,
third of patients presenting with acute cough. The estimated economic impact of non
influenza-related URIs is $40 billion annually.
Influenza epidemics occur every year between November and March in the
Northern Hemisphere. Approximately two thirds of those infected with influenza virus
exhibit clinical illness, 25 million seek health care, 100,000 to 200,000 require
hospitalization, and 40,000 to 60,000 die each year as a result of related complications.
The average cost of each influenza epidemic is $12 million, including the direct cost of
medical care and indirect cost resulting from lost work days. Pandemics in the 20th
century claimed the lives of more than 21 million people. A widespread H5N1 pandemic
in birds is ongoing, with threats of a human pandemic. It is projected that such a
pandemic would cost the United States $70 to $160 billion.
The reason why i chose this patient was that her case was the most interesting
among all the patients in the ward. There were a lot of problems that I could identify that
caught my interest and where we can give a lot of health teachings and interventions to
our client. In short, her case fits best in the criteria for choosing a case study because
her diagnosis was something a common one. I also want to go deeper with this kind of
case and learn more from it.
D. Definition of diagnosis
Upper respiratory tract infection (URI) is a nonspecific term used to describe acute
infections involving the nose, paranasal sinuses, pharynx, larynx, trachea, and bronchi.
The prototype is the illness known as the common cold, which is discussed here, in
addition to pharyngitis, sinusitis, and tracheobronchitis. Influenza is a systemic illness
that involves the upper respiratory tract and should be differentiated from other URIs.
Viruses cause most URIs, with rhinovirus, parainfluenza virus, coronavirus,
adenovirus, respiratory syncytial virus, Coxsackie virus, human metapneumovirus, and
influenza virus accounting for most cases. Group A beta-hemolytic streptococci
(GABHS) cause 5% to 10% of cases of pharyngitis in adults. Other less common
causes of bacterial pharyngitis include group C beta-hemolytic streptococci,
Corynebacterium diphtheriae, Neisseria gonorrhoeae, Arcanobacterium haemolyticum,
Chlamydophila (formerly Chlamydia) pneumoniae, Mycoplasma pneumoniae, and
herpes simplex virus. Streptococcus pneumoniae, Haemophilus influenzae, and
Moraxella catarrhalis are the most common organisms that cause the bacterial
superinfection of viral acute rhinosinusitis. Less than 10% of cases of acute
tracheobronchitis are caused by Bordetella pertussis, B. parapertussis, M. pneumoniae,
or C. pneumoniae.
Scope and Limitation
The case study merely covers data that have been gathered through interview per
assessment tool and chart referral on the day of the assessment phase in loading
assigned patients and in the succeeding days of the rotation, in the care formulated and
intervened to its progress as the weeks rotation ended. Thus, it is limited to the days in
the rotation the student nurse interacted with the client in the hope to gather the
necessary data to support the presentation which is not enough to acquire a bulk of
specific details.
E. Patients Profile
I.
HEALTH HISTORY
Patients Profile
Clients Name:
Mr. X
Age:
15 years old
Birthday:
12/15/1999
Address:
Civil Status:
Single
Sex:
Male
Nationality:
Filipino
Religion:
Roman Catholic
Height:
167 cm (52)
Weight:
78 kg
Admitting Physician:
Date of Admission:
Time of Admission:
1:21 PM
Chief Complaint:
Admitting Diagnosis:
The patients father was diagnosed with Renal Failure, and he also had
High blood pressure. While the patients mother was diagnosed with type 2
Diabetes. No other trace of underlying condition was reported by the patient.
with feverish
sensation and decrease appetite, at July 7, 2015 at 1:21 pm due to her chief complaints
of productive cough he was admitted at Davao Doctors Hospital and he was diagnosed
by Dr. Uy that she suffered with Upper Respiratory Tract Infection
Respiratory system
The Respiratory System is crucial to every human being. Without it, we would cease to
live outside of the womb. Let us begin by taking a look at the structure of the respiratory
system and how vital it is to life. During inhalation or exhalation air is pulled towards or
away from the lungs, by several cavities, tubes, and openings.
The organs of the respiratory system make sure that oxygen enters our bodies and
carbon dioxide leaves our bodies.
The respiratory tract is the path of air from the nose to the lungs. It is divided into two
sections: Upper Respiratory Tract and the Lower Respiratory Tract. Included in the
upper respiratory tract are the Nostrils, Nasal Cavities, Pharynx, Epiglottis, and the
Larynx. The lower respiratory tract consists of the Trachea, Bronchi, Bronchioles, and
the Lungs.
As air moves along the respiratory tract it is warmed, moistened and filtered.
Breathing and Lung Mechanics
Ventilation is the exchange of air between the external environment and the alveoli. Air
moves by bulk flow from an area of high pressure to low pressure. All pressures in the
7
respiratory system are relative to atmospheric pressure (760mmHg at sea level). Air will
move in or out of the lungs depending on the pressure in the alveoli. The body changes
the pressure in the alveoli by changing the volume of the lungs. As volume increases
pressure decreases and as volume decreases pressure increases. There are two
phases of ventilation; inspiration and expiration. During each phase the body changes
the lung dimensions to produce a flow of air either in or out of the lungs.
The body is able to stay at the dimensions of the lungs because of the relationship of
the lungs to the thoracic wall. Each lung is completely enclosed in a sac called the
pleural sac. Two structures contribute to the formation of this sac. The parietal pleura
are attached to the thoracic wall whereas the visceral pleura are attached to the lung
itself. In-between these two membranes is a thin layer of intrapleural fluid. The
intrapleural fluid completely surrounds the lungs and lubricates the two surfaces so that
they can slide across each other. Changing the pressure of this fluid also allows the
lungs and the thoracic wall to move together during normal breathing. Much the way
two glass slides with water in-between them are difficult to pull apart, such is the
relationship of the lungs to the thoracic wall.
The rhythm of ventilation is also controlled by the "Respiratory Centre" which is located
largely in the medulla oblongata of the brain stem. This is part of the autonomic system
and as such is not controlled voluntarily (one can increase or decrease breathing rate
voluntarily, but that involves a different part of the brain). While resting, the respiratory
center sends out action potentials that travel along the phrenic nerves into the
diaphragm and the external intercostal muscles of the rib cage, causing inhalation.
Relaxed exhalation occurs between impulses when the muscles relax. Normal adults
have a breathing rate of 12-20 respirations per minute.
The Pathway of Air
When one breathes air in at sea level, the inhalation is composed of different gases.
These gases and their quantities are Oxygen which makes up 21%, Nitrogen which is
78%, Carbon Dioxide with 0.04% and others with significantly smaller portions.
8
In the process of breathing, air enters into the nasal cavity through the nostrils and is
filtered by coarse hairs (vibrissae) and mucous that are found there. The vibrissae filter
macroparticles, which are particles of large size. Dust, pollen, smoke, and fine particles
are trapped in the mucous that lines the nasal cavities (hollow spaces within the bones
of the skull that warm, moisten, and filter the air). There are three bony projections
inside the nasal cavity. The superior, middle, and inferior nasal conchae. Air passes
between these conchae via the nasal meatuses.
Air then travels past the nasopharynx, oropharynx, and laryngopharynx, which are the
three portions that make up the pharynx. The pharynx is a funnel-shaped tube that
connects our nasal and oral cavities to the larynx. The tonsils which are part of the
lymphatic system, form a ring at the connection of the oral cavity and the pharynx.
Here, they protect against foreign invasion of antigens. Therefore the respiratory tract
aids the immune system through this protection. Then the air travels through the larynx.
The larynx closes at the epiglottis to prevent the passage of food or drink as a
protection to our trachea and lungs. The larynx is also our voicebox; it contains vocal
cords, in which it produces sound. Sound is produced from the vibration of the vocal
cords when air passes through them.
The trachea, which is also known as our windpipe, has ciliated cells and mucous
secreting cells lining it, and is held open by C-shaped cartilage rings. One of its
functions is similar to the larynx and nasal cavity, by way of protection from dust and
other particles. The dust will adhere to the sticky mucous and the cilia helps propel it
back up the trachea, to where it is either swallowed or coughed up. The mucociliary
escalator extends from the top of the trachea all the way down to the bronchioles, which
we will discuss later. Through the trachea, the air is now able to pass into the bronchi.
Inspiration
Inspiration is initiated by contraction of the diaphragm and in some cases the intercostal
muscles when they receive nervous impulses. During normal quiet breathing, the
phrenic nerves stimulate the diaphragm to contract and move downward into the
9
abdomen. This downward movement of the diaphragm enlarges the thorax. When
necessary, the intercostal muscles also increase the thorax by contacting and drawing
the ribs upward and outward.
As the diaphragm contracts inferiorly and thoracic muscles pull the chest wall
outwardly, the volume of the thoracic cavity increases. The lungs are held to the
thoracic wall by negative pressure in the pleural cavity, a very thin space filled with a
few milliliters of lubricating pleural fluid. The negative pressure in the pleural cavity is
enough to hold the lungs open in spite of the inherent elasticity of the tissue. Hence, as
the thoracic cavity increases in volume the lungs are pulled from all sides to expand,
causing a drop in the pressure (a partial vacuum) within the lung itself (but note that this
negative pressure is still not as great as the negative pressure within the pleural cavity-otherwise the lungs would pull away from the chest wall). Assuming the airway is open,
air from the external environment then follows its pressure gradient down and expands
the alveoli of the lungs, where gas exchange with the blood takes place. As long as
pressure within the alveoli is lower than atmospheric pressure air will continue to move
inwardly, but as soon as the pressure is stabilized air movement stops.
Expiration
During quiet breathing, expiration is normally a passive process and does not require
muscles to work (rather it is the result of the muscles relaxing). When the lungs are
stretched and expanded, stretch receptors within the alveoli send inhibitory nerve
impulses to the medulla oblongata, causing it to stop sending signals to the rib cage
and diaphragm to contract. The muscles of respiration and the lungs themselves are
elastic, so when the diaphragm and intercostal muscles relax there is an elastic recoil,
which creates a positive pressure (pressure in the lungs becomes greater than
atmospheric pressure), and air moves out of the lungs by flowing down its pressure
gradient.
10
Although the respiratory system is primarily under involuntary control, and regulated by
the medulla oblongata, we have some voluntary control over it also. This is due to the
higher brain function of the cerebral cortex.
When under physical or emotional stress, more frequent and deep breathing is needed,
and both inspiration and expiration will work as active processes. Additional muscles in
the rib cage forcefully contract and push air quickly out of the lungs. In addition to
deeper breathing, when coughing or sneezing we exhale forcibly. Our abdominal
muscles will contract suddenly (when there is an urge to cough or sneeze), raising the
abdominal pressure. The rapid increase in pressure pushes the relaxed diaphragm up
against the pleural cavity. This causes air to be forced out of the lungs.
Another function of the respiratory system is to sing and to speak. By exerting
conscious control over our breathing and regulating flow of air across the vocal cords
we are able to create and modify sounds.
Lung Compliance
Lung Compliance is the magnitude of the change in lung volume produced by a change
in pulmonary pressure. Compliance can be considered the opposite of stiffness. A low
lung compliance would mean that the lungs would need a greater than average change
in intrapleural pressure to change the volume of the lungs. High lung compliance would
indicate that little pressure difference in intrapleural pressure is needed to change the
volume of the lungs. More energy is required to breathe normally in a person with low
lung compliance. Persons with low lung compliance due to disease therefore tend to
take shallow breaths and breathe more frequently.
Determination of Lung Compliance Two major things determines lung compliance. The
first is the elasticity of the lung tissue. Any thickening of lung tissues due to disease will
decrease lung compliance. The second is surface tensions at air water interfaces in the
alveoli. The surface of the alveoli cells is moist. The attractive force, between the water
cells on the alveoli, is called surface tension. Thus, energy is required not only to
11
expand the tissues of the lung but also to overcome the surface tension of the water
that lines the alveoli.
To overcome the forces of surface tension, certain alveoli cells (Type II pneumocytes)
secrete a protein and lipid complex called ""Surfactant, which acts like a detergent by
disrupting the hydrogen bonding of water that lines the alveoli, hence decreasing
surface tension.
Upper Respiratory Tract
The upper respiratory tract consists of the nose and the pharynx. Its primary function is
to receive the air from the external environment and filter, warm, and humidify it before
it reaches the delicate lungs where gas exchange will occur.
Air enters through the nostrils of the nose and is partially filtered by the nose hairs, then
flows into the nasal cavity. The nasal cavity is lined with epithelial tissue, containing
blood vessels, which help warm the air; and secrete mucous, which further filters the
air. The endothelial lining of the nasal cavity also contains tiny hair like projections,
called cilia. The cilia serve to transport dust and other foreign particles, trapped in
mucous, to the back of the nasal cavity and to the pharynx. There the mucus is either
coughed out, or swallowed and digested by powerful stomach acids. After passing
through the nasal cavity, the air flows down the pharynx to the larynx.
Lower Respiratory Tract
The lower respiratory tract starts with the larynx, and includes the trachea, the two
bronchi that branch from the trachea, and the lungs themselves. This is where gas
exchange actually takes place.
1. Larynx
The larynx (plural larynges), colloquially known as the voice box, is an organ in our
neck involved in protection of the trachea and sound production. The larynx houses the
vocal cords, and is situated just below where the tract of the pharynx splits into the
12
trachea and the esophagus. The larynx contains two important structures: the epiglottis
and the vocal cords.
The epiglottis is a flap of cartilage located at the opening to the larynx. During
swallowing, the larynx (at the epiglottis and at the glottis) closes to prevent swallowed
material from entering the lungs; the larynx is also pulled upwards to assist this
process. Stimulation of the larynx by ingested matter produces a strong cough reflex to
protect the lungs. Note: choking occurs when the epiglottis fails to cover the trachea,
and food becomes lodged in our windpipe.
The vocal cords consist of two folds of connective tissue that stretch and vibrate when
air passes through them, causing vocalization. The length the vocal cords are stretched
determines what pitch the sound will have. The strength of expiration from the lungs
also contributes to the loudness of the sound. Our ability to have some voluntary control
over the respiratory system enables us to sing and to speak. In order for the larynx to
function and produce sound, we need air. That is why we can't talk when we're
swallowing.
1. Trachea
2. Bronchi
3. Lungs
The Right Primary Bronchus is the first portion we come to, it then branches off
into the Lobar (secondary) Bronchi, Segmental (tertiary) Bronchi, then to the
Bronchioles which have little cartilage and are lined by simple cuboidal
epithelium (See fig. 1). The bronchi are lined by pseudostratified columnar
epithelium. Objects will likely lodge here at the junction of the Carina and the
Right Primary Bronchus because of the vertical structure. Items have a tendency
to fall in it, whereas the Left Primary Bronchus has more of a curve to it which
would make it hard to have things lodge there.
13
The Left Primary Bronchus has the same setup as the right with the lobar,
segmental bronchi and the bronchioles.
The lungs are attached to the heart and trachea through structures that are
called the roots of the lungs. The roots of the lungs are the bronchi, pulmonary
vessels, bronchial vessels, lymphatic vessels, and nerves. These structures
enter and leave at the hilus of the lung which is "the depression in the medial
surface of a lung that forms the opening through which the bronchus, blood
vessels, and nerves pass" (medlineplus.gov).
A. Integumentary
GENERAL APPEARNCE
Patients skin colour has white skin complexion, no discoloration and skin texture
is smooth. Patient has good skin turgor as I pinched his skin and goes back
immediately in place. There was no presence of scaling noted. Hair is evenly
distributed, thin, short, black hair and no infestations noted. In patients finger
nails and toe nails, there was no problem deviations assessed.
14
Nutritional/metabolic Patterns
Height is 176cm and weight is 76kg which indicates within the ideal body weight.
Patient has good appetite and able to consume all food served.
BMI computation:
176cm/100= 1.76x2 = 3.52 (76kg/3.52= 21.59)
Body Mass Index Ranges
Genitourinary
Musculoskeletal
Upon assessment with the patients gait, posture weight bearing stance, there
are no abnormalities noted. No decrease of ROM, tenderness and misalignment noted.
Neurological System
Patient is responsive, able to interact and socialize with other people, there was
no change in level of consciousness, and vocalizes sounds.
Cranial Nerve Function
CN 1-Olfactory
CN VI-Trigeminal
CN VII-Facial
CN VIII-Acoustic
CN IX-Glossopharyngeal
CN X-Vagus
CN XI-Spinal accessory
CN XII-Hypoglossal
Sensory Function
Touch and Pain were intact.
Reflexes
Reflexes are normal, visible muscle twitch and extension of lower leg.
Etiology
Upper respiratory infection is generally caused by the direct invasion of the inner
lining (mucosa or mucus membrane) of the upper airway by the culprit virus or bacteria.
In order for the pathogens (viruses and bacteria) to invade the mucus membrane of the
upper airways, they have to fight through several physical and immunologic barriers.
The hair in the lining of the nose acts as physical barrier and can potentially trap the
invading organisms. Additionally, the wet mucus inside the nasal cavity can engulf the
viruses and bacteria that enter the upper airways. There are also small hair-like
structures (cilia) that line the trachea which constantly move any foreign invaders up
towards the pharynx to be eventually swallowed into the digestive tract and into the
stomach. In addition to these intense physical barriers in the upper respiratory tract, the
immune system also does its part to fight the invasion of the pathogens or microbes
entering the upper airway. Adenoids and tonsils located in the upper respiratory tract
are a part of the immune system that helps fight infections. Through the actions of the
17
specialized cells, antibodies, and chemicals within these lymph nodes, invading
microbes are engulfed within them and are eventually destroyed.
18
H. Pathophysiology
19
Narrative Report
Predisposing
Precipitating
Computer gamer
15 years old
Secondhand smoker
Allergy rhinitis
Sedentary lifestyle
20
Doctors Order
Rationale of Order
service of Doctor Yu
health service
>LABS:
-CBC
-U/A
-CXR (PA)
MEDs
1. Ambrolex OD 75 mg/ PO
2. Brompheniramine
( Nasatapp) 1 tab BID
3. Omeprazole
>V/S q 4o
>I&O q shift
>Pls inform AP
>refer accordingly
7/7/2015
Laboratory Results
7/6/2015
Urine Analysis
Color
Result
Normal Values
Rationale
Yellow
Normal
23
Negative
Glucose
Negative
Normal
Transparency
Clear
Negative
Normal
Protein
Negative
1.002-1.030
Normal
Specific Gravity
1.015
Normal
0-4/hpf
Microscopic Exam:
Pus Cells
1-3/hpf
RBC
0-2/hpf
0-3/hpf
Normal
Normal
Amourphous Urates /
Phosphates
Epithelial
Few
Squamous
Cells
Negative
Few
Bacteria
Occasional
Indicates Infection
7-6-2015
Complete Blood Count
Result
Normal values
Implication
HCT
40.1
37-47vol %
Normal
HGB
12.9
12-16gms %
Normal
WBC
8,200
5,000-10,000/mm3
24
Normal
PLATELET
310,000
150,000-400,000/mm3
69
50-62 %
Normal
Diff. Count
Neutrophils
Respond to any
inflammation
Granulocyte
50
43.4-76.2 %
Lymphocytes
43
17.4-48.2 %
Monocytes
4.5-10.5 %
25
Normal
Normal
Normal
Generic
name/Brand
Generic
name
/brand name
Ambrolex
Classificati
Dose/
Mechanism
Specific
Contra-
on
route
of Action
indication
indication
Mechanism
Specific
Contra-
indication
Acute and
indication
There
Classificatio Freque
Dose/
n
antibiotic
route
ncy
of Action
OD 75 Concentratio
Freque
mg PO n of
ncy
antibiotics
Adverse reaction
Nursing Precaution
Adverse reaction
Nursing Precaution
Occasional
Observe
chronic
are
disorder of
absolute
when given
the
contraindicatio
concomitantl
respiratory
ns
y.
tract
patients
associated
gastric
with
ulceration
pathological
relative
ly thickened
caution should
mucus and
be observed.
but
in mild.
with
if
rate
and
baseline
data.
taking
other
medications.
It is advisable to avoid
use
impaired
mucus
transport.
26
during
the
first
trimester of pregnancy.
Salbuterol
bronchodila
tor
1 neb
Stimulates
Relief of
Hypersensitivit
-Assess cardio-
Beta 2
bronchospa
y to
receptors of
sm in
salbutamol,
palpitations,
bronchioles
bronchial
also to
tachycardia,
by increasing
asthma,
atropine and
hypertension,
levels which
chronic
its derivatives.
headache, nausea,
relaxes
bronchitis,
Threatened
vomiting, dizziness,
smooth
emphysem
abortion
hyperactivity,
muscles to
a and other
during first or
insomnia,
produce
reversible
second
hypotension,
bronchodilati
obstructive
trimester.
heartburn, epistaxis,
paradoxical
on. Also
pulmonary
cough
bronchospasm
cause CNS
disease
stimulation,
cardiac
stimulation,
increase in
diuresis,
skeletal
muscle
tremors and
increase
27
-Determine history of
previous meds and
ability to self-medicate.
gastric acid
secretions.
28
Generic
Classificatio
Dose/
Mechanism
Specific
Contra-
Adverse reaction
/brand name
route
of Action
indication
indication
Nursing Precaution
Freque
ncy
Omeprazole
Antisecretor
40mg
Gastric acid-
Reduction
Contraindicate
Headache, dizziness,
-Arrange regular
IVTT
pump
of risk of
d with
asthenia, vertigo,
medical check-ups.
inhibitor:
upper GI
hypersensitivit
insomnia, apathy,
-Advise pt to report
pump
Suppresses
bleeding
y to
anxiety, paresthesias,
inhibitor
gastric acid
secretion by
its
rash, inflammation,
specific
components.
urticaria, pruritus,
Proton
OD
inhibition of
the
diarrhea, abdominal
hydrogen-
pain, nausea,
potassium
vomiting,
ATPase
constipation, dry
enzyme
mouth
system at the
secretory
surface of
the gastric
parietal cells;
29
effects.
blocks the
final step of
acid
production.
Generic
Classificatio
Dose/
Mechanism
Specific
Contra-
Adverse reaction
/brand name
route
of Action
indication
indication
Freque
ncy
30
Nursing Precaution
Paracetamol Antipyretic
PRN
Inhibits the
>Mild pain
Hypersensitivit
Hema: hemolytic
~ Advise parents or
for
synthesis of
>Fever
y to
anemia,
caregivers to check
acetaminophe
neutropenia,
concentrations of liquid
s that may
n or
leukopenia,
preparations. Errors
serve as
phenacetin;
pancytopenia.
mediators of
use with
Hepa: jaundice
liver damage.
pain and
alcohol.
Metabolic: hypoG
fever,
GI: HEPATIC
presence of associated
primarily in
FAILURE,
signs (diaphoresis,
the CNS
HEPATOTOXICITY
tachycardia, and
(overdose)GU: renal
malaise).
failure (high
doses/chronic use).
acetaminophen longer
fever q prostaglandin
4 hrs
BRAND NAME
Acetylcysteine
GENERIC NAME
Exflem
Mucolytic
Reference :
MECHANISM OF
ADVERSE
SIDE EFFECTS
ACTION
Acetylcysteine
REACTION
> fever
> drowsiness
> tachycardia
fever, rhinorrhea,
32
DOSAGE
NURSING
Usual:
RESPONSIBILITIES
>Assess type,
frequency, and
60 mg 1 tab in
characteristics of
free sulfhydryl
> dyspnea
drowsiness, clamminess,
glass of
patients cough
> rash
water
edition
> chills
broncho constriction
>Monitor patient
fortachycardia
viscosity. This
action increases
with increasing pH
aspiration of excess
and is most
significant at pH 7
Bronchospasm
to 9. The mucolytic
Actual:
(unpredictable);
action of
acetylcysteine is not
of H2O OD @hs
notify physician
affected by the
immediately if either
presence of DNA.
occurs.
>Instruct patient to
notify prescribe
immediately about
nausea, rash or
vomiting
33
Assessment
Diagnosis
Planning
Interventions
34
Rationale
Evaluation
Subjective:
Objective:
Conscious/cohere
After 8 hours of
clearance r/t
continues nsg.
increased
Interventions the
production of
bronchial
maintain airway
secretions as
patency
manifested by
nt
Body malaise
Productive cough
Wheezes upon
(yellow to green
secretions
Productive cough
and coughing
Restlessness
(yellow to green
exercise.
noted
sputum
Discomfort noted
Restlessness
Facial Grimace
Chest pain
noted
Discomfort
Facial Grimace
Monitor Vital
signs
fowlers position
To facilitate
Improves
mobilizing
secretions w/o
coughing exercise
causing fatigue
Avoid exposure to
To avoid allergic
reaction
cigarette smoke,
form dyspnea.
expectorate
helps in
breathing and
Verbalized relief
baseline data
ventilation and
irritants such as
expansion
to do proper deep
Serves as
maximum lung
Learn and
perform breathing
Expectorate
auscultation
sputum
Ineffective airway
aerosol and
fumes
Auscultate breath
sounds
Increase fluid
intake
To ascertain
progress
Helps liquefy
secretions
35
Suction as
To clear airway
ordered
Provide adequate
Provide oxygen
inhalation as
amount of oxygen
ordered
secretions for
Administer
easy expulsion.
medication as
ordered
36
Assessment
Subjective:
Diagnosis
Conscious/cohere
nt
Warm to touch
noted
Flushed face
noted
Febrile with a
After 8 hours of
continuous TSB,
the pt.s
temperature as
temperature will
manifested by
decrease from
Warm to touch
37.9 to 37C
Flushed face
Febrile with a
verbalized
thermoregulation
Gitugnaw ko as
Objective:
Ineffective
Planning
Interventions
Monitor VS
Increase fluid
intake
The patient
baseline data
temperature is
fluctuating, goal
core temperature
partially met.
To decrease
produce heat
Facilitate comfort
Facilitate heat
temperature of
38.2C
Provide sufficient
loss by means of
clothing
evaporation
Perform TSB
Helps lower
temperature
Administer
within normal
antipyretics as
range
37
Evaluation
Serves as
metabolism that
temperature of
37.9C
Rationale
ordered
38
Assessment
Subjective:
dili ko ganahan
mo kaon
Objective:
Refusal to eat
Poor muscle
tonicity
Body weakness
Diagnosis
Altered nutrition
Planning
Interventions
Evaluation
After 4 hours of
nursing
Weight on regular
Monitor nutritional
requirements R/T
interventions,
state and
was able to
loss of appetite as
patients appetite
effectiveness of
understand the
evidenced by
will be improved:
interventions
importance of
dysfunctional
from 2
To appeal to client
eating pattern.
tablespoons to at
basis
Discuss eating
least 5
habits including
tablespoons per
food preferences.
meal.
Serve favourite
To stimulate the
appetite
noted
Rationale
contraindicated.
Restlessness
To stimulate the
appetite
attractive.
Prevent and
minimize
39
May have
unpleasant
negative effect on
odours.
appetite/eating
Emphasize the
importance of
well-balanced
nutrition diet
40
Promote wellness
41