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After an action potential, the membrane potential dips under the resting potential. After
this happens, what actions of the sodium-potassium pump cause the membrane potential
to increase so that the resting potential is restored?
1. Chloride leaving the cell
2. Calcium entering the cell and chloride leaving the cell
3. Sodium leaving the cell
4. Sodium leaving the cell and potassium entering the cell
5. 2 and 4
After an action potential travels down the axon of a presynaptic neuron and causes the
intake of calcium ions through calcium-gated ion channels in the synaptic terminal, what
is the immediate effect of this?
1. The postsynaptic neuron fires an action potential
2. The presynaptic neuron generates another action potential
3. The synaptic vesicles of the presynaptic neuron fuse with the cell membrane and
release neurotransmitters into the synapse to communicate with the postsynaptic
neuron
4. Ligand-gated ion channels open on the post-synaptic neuron
5. The presynaptic neuron releases chloride ions into the synapse
The process described in the answer to question # 3 is called what?
1. Exocytosis
2. Reuptake
3. Concentration gradient
4. Depolarization
5. Excitatory Postsynaptic Potential
Which of the following is not correct?
1. The rising phase of the action potential (depolarization) is caused by sodium
entering the cell.
2. At rest, the inside of the neuron is more negative with respect to the outside
3. The speed of axonal conduction (action potential propagation) is faster in
myelinated axons.
4. At rest, there is more potassium inside of the cell, and more sodium inside.
5. The amplitude of an action potential depends on the intensity of the stimulus that
caused it.
In the resting neuron, potassium ion channels are by nature a bit leaky, and so some
potassium ions will the leak out of through these channels due to the concentration
gradient. If this stands uncorrected for long enough, the resting potential cant be
maintained. What corrects this problem?