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  • Pathophysiology CHF Mine

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    Calimlim Kim
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    This document outlines the pathophysiology of heart failure. It describes how non-modifiable risk factors like family history and age, along with modifiable risk factors like an unhealthy diet, smoking, and alcohol can lead to the buildup of cholesterol in artery walls and the development of atherosclerosis. Over time, this narrowing of blood vessels reduces blood flow to the heart and body, causing the heart to work harder and become less efficient. This increased workload stresses the heart and can eventually lead to congestive heart failure if left untreated.

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    Patho Physio

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    This document outlines the pathophysiology of heart failure. It describes how non-modifiable risk factors like family history and age, along with modifiable risk factors like an unhealthy diet, smoking, and alcohol can lead to the buildup of cholesterol in artery walls and the development of atherosclerosis. Over time, this narrowing of blood vessels reduces blood flow to the heart and body, causing the heart to work harder and become less efficient. This increased workload stresses the heart and can eventually lead to congestive heart failure if left untreated.

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    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    218 views2 pages

    Pathophysiology CHF Mine

    Uploaded by

    Calimlim Kim
    This document outlines the pathophysiology of heart failure. It describes how non-modifiable risk factors like family history and age, along with modifiable risk factors like an unhealthy diet, smoking, and alcohol can lead to the buildup of cholesterol in artery walls and the development of atherosclerosis. Over time, this narrowing of blood vessels reduces blood flow to the heart and body, causing the heart to work harder and become less efficient. This increased workload stresses the heart and can eventually lead to congestive heart failure if left untreated.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    of 2

    PATHOPHYSIOLOGY

    Modifiable Non-modifiable
    *Diet *Family history of hpn,
    -high fat, high sodium heart problems
    *alcohol
    *smoking *Age:76 yrs. old

    Bad cholesterol (LDL) congest into the wall of


    arteries

    Activation of specialized cells

    Theyll become enlarge cholesterol and rich cell


    (film cells)

    Invasion to the vessel wall

    Atherosclerosis

    Increase size of the plaque

    Narrowing of the vessels

    vasoconstriction
    Decrease oxygenated blood flowing into the heart
    Hypertension muscle

    Decrease heart contraction

    Decrease blood flow Reduces efficiency of myocardium through


    all over the body damage and overloading

    Activation of RAAS
    Decrease oxygen supply Increase systemic vascular
    and demand resistance
    The kidney will
    release Renin
    Chest pain Stimulation of adrenergic
    system
    Angiotensinogen
    Increase force of left
    ventricle contraction
    Angiotensin I
    Increase left ventricle
    workload
    Angiotensin II (ACE)

    Aldosterone Increase left ventricle


    oxygen demand
    Increase sodium
    Left ventricle hypoxia
    Water retention
    Pulmonary edema

    Bipedal Edema Pulmonary vascular


    resistance
    Right ventricular failure

    Activity Intolerance Weakness and Decrease cardiac output


    Easy fatigability
    Ventricular remodelling

    Progressive loss of
    cardiac output

    Myocardial hypertrophy

    Loss of myocytes

    Congestive heart failure

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