 Who came up with Sympatheticatonia: Korr

 Etiology of instability: trauma, congenital changes, postural insults

 Other theories associated with VSC Phase 2: VBI & Instability
 Four components of the Sandoz theory of immobilization: articular overstress, insufficiency and
instability, episodic fixation,
 Structural anomaly that causes instability: Down’s, retrolisthesis
 VSC phase of instability: Phase 2
 Prolonged immobilization leads to: permeant impairment or stabilization
 Name the three phases of the Kirkaldy Willis model: Dysfunction, Unstable, Stabilization
 VSC phase associated with Immobilization Degeneration: Phase 3 (Sandoz-phase 4)
 Instability confirmed by: Stress x-ray
 Use it or: lose it
 2 functional stabilizing systems of the spine: local and regional
Local: multifidus, rotat, intertransverse
Regional: abdominals, QL, intercostalis,
 The motion that decreases the fatigue of intrinsic muscles and lowers threshold of buckling:
vibration
 Primary proponents of Spinal buckling: Triono
 Deformity occurs with prolong static posture: Creep
 Can alter the dynamics of a buckled spinal region: Adjustment
 Neuroimmune AKA: neurodystrophy, neurodystrophic
 Chiropractic technique associated with myelopathy: HIO/ UC
 Type of herniation is associated with myelopathy: Central
 Transport system for the proteins: Macromolecule transport
 Prolonged bombardment of the dorsal horn will lead to: sensitization/peripheral modulization
 Age group most associated with having VBI: 31-35 OR younger than 45
 Neuropathology is closely associated with this theory: AXT
 The procedure if a patient presents with s/s of a VBI post adjustment: STOP, OBSERVE, REFER
 Primary indicator of SDF in kids: torticollis
 This will be demonstrated with chronic neuropathology: anesthesia
 S/s associated with myelopathy: UMNL, LMNL @ site of lesion
 Persons associated with myelopathy: BJ
 The change noted in action potentials with nerves demonstrating neuropathology: decreased AP
 This hormone is increased during neuroimmune hypothesis: Cortisol
 Degeneration associated with AXT aberration: Wallerian
 S/S associated with AXT aberration: Pain moves
 Three stages of GAS: Alarm, Resistance, Exhaustion/Adaptation
 Lab finding associated with Neuroimmune hypothesis: IgG
 Hormone associated with AXT: NGF
 S/S neuropathology: LMNL
 Person with neuroimmune: Hans Selye
 Primary person with neuropathology: DD Palmer