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Insider CLINICAL
The following notes are based upon the widely
known Jam’ El-Majam’ clinical series, So you will find Part II
nothing new except for organization and
simplification. The aim is to offer it in a more Cardiology sheet
concise way easily to remember and handle. That is E x a m - o r i e n t e d
to say it’s the “Skimmed Jam’ El-Majam’”.
Internal Medicine | Cardiology sheet Internal Medicine | Cardiology sheet
1
Examine the Heart: II. Cardiac:
I. Inspection AND Palpation: - Dextrocardia.
- RVH → Outwards; LVH → Downwards AND outwards.
1. Pericardial bulge:
III. Chest:
Congenital HD; Rheumatic HD.
Pericardial effusion; RVH. - PE; Pneumothorax → Push; Fibrosis; Collapse → Pull.
I.
2. Apex: IV. Abdominal:
Site: - ↑ IAP (Ascites; Pregnancy) → Shifted up.
- Normally in left 5th ICS just inside MCL.
Extent: V. Misc.→ Kyphoscoliosis.
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- Localized → Normally AND LVH.
- Diffuse → RVH. Difference () RVH AND LVH:
- Double → MI.
LVH RVH
- Bifid → BBB.
Apex:
Character: Normally no special character.
Thrill : Only low frequency murmurs are palpable. Site Downwards AND outwards. More outwards.
Relation to systole: Retraction OR Bulge. Extent Localized. Diffuse.
3. Pulsations. Character 1. Volume overload: Slapping.
4. Thrill. Hyperdynamic.
5. State of the skin: 2. Pressure over:
Dilated vessels (SVC Thrombosis); Pigmentation; Scar. Heaving.
II. Percussion. Relation Bulge Retraction
to systole (Parasternal retraction) (Parasternal bulge)
III. Auscultation:
Misc.:
For Heart sounds; Added sounds; Murmurs; Pericardial rub.
Pulsation 1. Epigastric pulsation.
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2. Parasternal pulsation:
Cause of Abnormal site of the apex: If RVE + LAE:
-
I. Physiological: - Uplift pulsation.
If RVE only:
- Long thin person : 6th ICS.
- Heaving pulsation.
- Children > 2 years : 4th ICS.
Dullness 1. Lower ⅓ of sternum.
- During inspiration : More down. -
2. Widened bare area.
- Position : More down in sitting; laterally in left side.
Internal Medicine | Cardiology sheet Internal Medicine | Cardiology sheet
2
Causes of Absent (Invisible OR Not palpable) Apex: Causes of Dullness:
1. Obesity. (Thick chest wall) I. Pulmonary area (2nd left ICS):
2. Behind a rib. - Pulmonary artery dilation; LAE.
3. Pleural effusion; Pericardial effusion; Emphysema. - Aortic artery aneurysm.
4. Advanced HF. (weak) II. Aortic area (2nd right ICS):
5. Dextrocardia. - Ascending aortic artery dilation.
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III. Cardiac waist (3rd left ICS):
Causes of Pulsations: - LAE.
I. Suprasternal pulsation: IV. Right of the sternum:
- Short obese with short neck and High diaphragm. - RAE; Huge LAE.
- Hyperdynamic cir. (AR) V. Outside the apex:
- Coarctation of the aorta.
- Pericardial effusion; Ventricular aneurysm.
- High Aortic Arch.
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- Aneurysm of aortic arch.
II. Epigastric pulsation: Causes of ↓ Bare area:
- RVH. 1. Pneumothorax.
- Hepatic pulsations → TR (Systolic); TR AND TS (Diastolic). 2. Emphysema.
- Abdominal Aorta. ــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــ
III. Pulsation to right of the sternum: (Fingers Tips) Causes of ↑ Bare area:
- RAE; Huge LAE. 1. RVE.
- Aortic Aneurysm.
2. Pericardial effusion.
- Internal mammary artery in lactating female.
3. Fibrosis; Collapse.
IV. Parasternal pulsation: (By Paroxysmal part of a palm)
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- RVH → Uplift OR Heaving.
Causes of Huge (Bovine) heart:
V. Pulmonary area pulsation: (Fingers Tips; Left hand ulnar border)
- Pulmonary artery dilation or aneurysm; LAE. 1. Multivalvular disease.
VI. Aortic area pulsation: (Fingers Tips) 2. Pericardial effusion.
3. Dilated cardIomyopathy.
- Aortic artery dilation.
4. Long standing hypertensive HF.
Internal Medicine | Cardiology sheet Internal Medicine | Cardiology sheet
3
Causes of Small heart: Difference () Wide and Reversed splitted S2:
1. COPD. Wide splitted S2 Reversed splitted S2
2. Addison’s disease; Senile cardiac atrophy. Haemodynamics Delayed closure of Delayed closure of Aortic
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Causes of Accentuated AND Weak S1: Causes ASD. PDA.
PS. AS.
Accentuated S1 Weak S1 RBBB. LBBB.
MS. 1. Loss of valvular component: Effect of deep ↑ Disappear
Hyperdynamic circulation. MR. inspiration
Tachycardia. Calcified MS.
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Exercise; Emotion.
2. Loss of Muscular component:
Children.
Myocarditis.
Causes of Single S2:
Thin chest wall.
Cardiomyopathy. 1. TOF; Pulmonary atresia.
3. Misc.: 2. Truncus arteriosus; Very large VSD.
Bradycardia. ــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــ
Absent apex.
Causes of S3 (Protodiastolic sound):
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lateral position
↑ in left
Blowing AT Apex heart base
With propagation to
[in Pt. leaflet Innocent murmurs:
Murmur which is
regurge]
- Early systolic soft murmurs.
MS Mid- Localized - Best heard over P area AND Left sternal edge.
Rumbling -
Diastolic TO Apex
- ↑ with standing and respiration.
AR Max. Apex - Causes: Hyperdynamic circulation; Minimal organic lesions; Chest
OVER A1
↑ with leaning
Early Soft Neck deformities.
OR Max.
forward
Diastolic Blowing ــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــــ
OVER A2
[in Rh. AR]
AS Ejection Max. Apex
Harsh OVER A1
Systolic Neck
The End
TR Max. Apex Part II: Cardiology sheet
Soft
Pansystolic OVER Clinical sheets of Internal medicine
Blowing
TS Max. -
Murmur which is
PS Max. Apex
Ejection
Harsh OVER Neck
Systolic
P Area
NB:
- Severity of the lesion depends on the duration (which is determined by
pressure gradient) NOT the intensity of the murmur.