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ELECTRO CARDIOGRAM
ECG, or EKG [from the German Elektrokardiogramm]) is a transthoracic interpretation of the electrical activity of the heart over time captured and externally recorded by skin electrodes. It is a noninvasive recording produced by an electrocardiographic device.
ECG Grid
ECG Grid Left to Right = Time/duration Vertical measure of voltage (amplitude) Expressed in mm
P-Wave
Depolarization of atrial muscle Low voltage - 2-3mm in amplitude Duration < 0.11 seconds ( 2 to 2.5 small squares)
PR interval
Duration is 0.12 to 0.20 s (3 - 5 small squares) . Time taken for the excitation to spread from the SA node through the atrium and AV node down the bundle of HIS to the ventricular muscle.
QRS Complex
Depolarization of ventricles which has larger muscle mass. Amplitude as high as 25mm. Duration with Normal Conduction < 0.12 ( 3 small squares). J point is where the S wave (or whatever the last wave in the QRS is) ends and the ST segment begins.
QRS Complex
ST Segment
Time between completion of depolarization and onset of repolarization Normally isoelectric & gently blends into upslope of T wave Point where ST takes off from QRS= J point Plays important role in diagnosis of ischemic heart disease
T - Wave
Repolarization of the ventricles. Tall tented T waves could represent hyperkalaemia. T wave inversion can represent coronary ischaemia, previous infarction or electrolyte abnormality such as hypokalaemia.
QT Interval
Beginning of QRS to end of T Wave. Normal variations with HR and gender Abnormalities. Prolonged commonly from drugs like Procaine or Quinidine or electrolyte imbalance. Increased opportunity for R on T, ventricular re-entry rhythms and sudden death,
Einthoven triangle
Pioneered by Willam Einthoven Machine that was sensitive enough to reliably measure electrical differences between two different parts of the body
V1, V2
V3, V4
Septal
Anterior
V5, V6
V1 to V4 V3 to V6 LI , aVL LII , L III , aVF
Lateral
antero-septal Antero-lateral High lateral Inferior
Dysrhythmias
Dysrhythmias are disorders of the formation or conduction (or both) of the electrical impulse within the heart. These disorders can cause disturbances of the heart rate, the heart rhythm, or both.
CLASSIFICATION OF DYSRYTHMIAS
Atrial Dysrythmias
Junctional dysrhythmias
Atrio-ventricular dysrhythmias Ventricular dysrhythmias
Heart blocks
SADS
I. Atrial dysrhythmias
Premature Atrial Contractions (PACs) Wandering Atrial Pacemaker Multifocal atrial tachycardia Atrial flutter Atrial fibrillation (Afib)
entry circuit crosses between the atria and ventricles somewhere other than the AV node. Wolff-Parkinson-White syndrome
Lown-Ganong-Levine syndrome
Cont. Accelerated idioventricular rhythm Monomorphic Ventricular tachycardia Polymorphic ventricular tachycardia Ventricular fibrillation
V.
Heart blocks :-
Heart blocks
These are also known as AV blocks, because the vast majority of them arise from pathology at the atrioventricular node. They are the most common causes of bradycardia
SADS is a term used to describe sudden death due to cardiac arrest brought on by an arrhythmia in the absence of any structural heart disease.
Sinus bradycardia
Appears like normal sinus rhythm except that the rate is slower than 60 per minute Athletes
Sinus Tachycardia
normal sinus rhythm except that the rate is faster than 100 per minute.
Sinus arrhythmia
Similar to normal sinus rhythm except that it the rate is irregular Relatively common in young and is often asymptomatic
Atrial dysrythmias