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An intensive care unit (ICU), also sometimes known as a critical care unit or an
intensive therapy department is a special ward found inside most hospitals. It
provides intensive care (treatment and monitoring) for people who are in a critically
ill or unstable condition. Patients in ICUs need constant medical support to keep
their body functions going. They may not be able to breathe on their own, and
may have multiple organ failure, so medical equipment takes the place of these
functions while they recover.There are several circumstances in which a person may
be admitted to intensive care, for example, following surgery, or after an accident or
severe illness. ICU beds are a very expensive and limited resource because they
provide specialized monitoring equipment, a high degree of medical expertise and
constant access to highly trained nurses (usually one nurse for each bed). Being in an
ICU can be a daunting experience both for the patient and his or her friends and
family. The healthcare professionals in ICUs understand this and are there to help and
support both patients and their families during their time in intensive care.
Myocardial infarction (MI) is the irreversible necrosis of heart muscle
secondary to prolonged ischemia. This usually results from an imbalance of
oxygen supply and demand. The appearance of cardiac enzymes in the
circulation generally indicates myocardial necrosis. MI is considered, more
appropriately, part of a spectrum referred to an acute coronary syndromes
(ACSs), which also includes unstable angina and non–ST-elevation MI
(NSTEMI). Patients with ischemic discomfort may or may not have ST-
segment elevation. Most of those with ST-segment elevation will develop Q
waves. Those without ST elevations will ultimately be diagnosed with
unstable angina or NSTEMI based on the presence of cardiac enzymes. MI
may lead to impairment of systolic function or diastolic function and to
increased predisposition to arrhythmias and other long-term complications.
b. General Objective
1. Describe Critical Care as a collaborative, holistic
approach that includes the patient, family and significant
others
Diagnosis: Nosocomial Pneumonia; CAD, ACS, NSTMI, Killip II, HCVD, FC II,
Intracerebral he, (L) Basal Ganglia with intraventricular extension
1. Right Coronary
2. Left Anterior Descending
3. Left Circumflex
4. Superior Vena Cava
5. Inferior Vena Cava
6. Aorta
7. Pulmonary Artery
8. Pulmonary Vein
9. Right Atrium
10. Right Ventricle
11. Left Atrium
12. Left Ventricle
13. Papillary Muscles
14. Chordae Tendineae
15. Tricuspid Valve
16. Mitral Valve
Coronary Arteries. Because the heart is composed primarily of
cardiac muscle tissue that continuously contracts and relaxes, it
must have a constant supply of oxygen and nutrients. The
coronary arteries are the network of blood vessels that carry
oxygen- and nutrient-rich blood to the cardiac muscle tissue. The
blood leaving the left ventricle exits through the aorta, the body’s
main artery. Two coronary arteries, referred to as the "left" and
"right" coronary arteries, emerge from the beginning of the aorta,
near the top of the heart. The initial segment of the left coronary
artery is called the left main coronary. This blood vessel is
approximately the width of a soda straw and is less than an inch
long. It branches into two slightly smaller arteries: the left
anterior descending coronary artery and the left circumflex
coronary artery. The left anterior descending coronary artery is
embedded in the surface of the front side of the heart. The left
circumflex coronary artery circles around the left side of the heart
and is embedded in the surface of the back of the heart. Just like
branches on a tree, the coronary arteries branch into
progressively smaller vessels. The larger vessels travel along the
surface of the heart; however, the smaller branches penetrate the
heart muscle. The smallest branches, called capillaries, are so
narrow that the red blood cells must travel in single file. In the
capillaries, the red blood cells provide oxygen and nutrients to the
cardiac muscle tissue and bond with carbon dioxide and other
metabolic waste products, taking them away from the heart for
disposal through the lungs, kidneys and liver. When cholesterol
plaque accumulates to the point of blocking the flow of blood
through a coronary artery, the cardiac muscle tissue fed by the
coronary artery beyond the point of the blockage is deprived of
oxygen and nutrients. This area of cardiac muscle tissue ceases to
function properly. The condition when a coronary artery becomes
blocked causing damage to the cardiac muscle tissue it serves is
Aorta. The aorta is the largest single blood vessel in the body. It is approximately the diameter
of your thumb. This vessel carries oxygen-rich blood from the left ventricle to the various
parts of the body.
Pulmonary Artery. The pulmonary artery is the vessel transporting de-oxygenated blood from
the right ventricle to the lungs. A common misconception is that all arteries carry oxygen-rich
blood. It is more appropriate to classify arteries as vessels carrying blood away from the heart.
Pulmonary Vein. The pulmonary vein is the vessel transporting oxygen-rich blood from the
lungs to the left atrium. A common misconception is that all veins carry de-oxygenated blood.
It is more appropriate to classify veins as vessels carrying blood to the heart.
Right Atrium. The right atrium receives de-oxygenated blood from the body through the
superior vena cava (head and upper body) and inferior vena cava (legs and lower torso). The
sinoatrial node sends an impulse that causes the cardiac muscle tissue of the atrium to contract
in a coordinated, wave-like manner. The tricuspid valve, which separates the right atrium from
the right ventricle, opens to allow the de-oxygenated blood collected in the right atrium to
flow into the right ventricle.
Right Ventricle. The right ventricle receives de-oxygenated blood as the right atrium contracts.
The pulmonary valve leading into the pulmonary artery is closed, allowing the ventricle to fill
with blood. Once the ventricles are full, they contract. As the right ventricle contracts, the
tricuspid valve closes and the pulmonary valve opens. The closure of the tricuspid valve
prevents blood from backing into the right atrium and the opening of the pulmonary valve
allows the blood to flow into the pulmonary artery toward the lungs.
Left Atrium. The left atrium receives oxygenated blood from the lungs through the pulmonary
vein. As the contraction triggered by the sinoatrial node progresses through the atria, the
blood passes through the mitral valve into the left ventricle.
Left Ventricle. The left ventricle receives oxygenated blood as the left atrium contracts. The
blood passes through the mitral valve into the left ventricle. The aortic valve leading into the
aorta is closed, allowing the ventricle to fill with blood. Once the ventricles are full, they
contract. As the left ventricle contracts, the mitral valve closes and the aortic valve opens. The
closure of the mitral valve prevents blood from backing into the left atrium and the opening of
the aortic valve allows the blood to flow into the aorta and flow throughout the body.
Papillary Muscles. The papillary muscles attach to the lower portion of the interior
wall of the ventricles. They connect to the chordae tendineae, which attach to the
tricuspid valve in the right ventricle and the mitral valve in the left ventricle. The
contraction of the papillary muscles opens these valves. When the papillary
muscles relax, the valves close.
Chordae Tendineae. The chordae tendineae are tendons linking the papillary
muscles to the tricuspid valve in the right ventricle and the mitral valve in the left
ventricle. As the papillary muscles contract and relax, the chordae tendineae
transmit the resulting increase and decrease in tension to the respective valves,
causing them to open and close. The chordae tendineae are string-like in
appearance and are sometimes referred to as "heart strings."
Tricuspid Valve. The tricuspid valve separates the right atrium from the right
ventricle. It opens to allow the de-oxygenated blood collected in the right atrium to
flow into the right ventricle. It closes as the right ventricle contracts, preventing
blood from returning to the right atrium; thereby, forcing it to exit through the
pulmonary valve into the pulmonary artery.
Mitral Value. The mitral valve separates the left atrium from the left ventricle. It
opens to allow the oxygenated blood collected in the left atrium to flow into the
left ventricle. It closes as the left ventricle contracts, preventing blood from
returning to the left atrium; thereby, forcing it to exit through the aortic valve into
the aorta.
Pulmonary Valve. The pulmonary valve separates the right ventricle from the
pulmonary artery. As the ventricles contract, it opens to allow the de-oxygenated
blood collected in the right ventricle to flow to the lungs. It closes as the ventricles
relax, preventing blood from returning to the heart.
Aortic Valve. The aortic valve separates the left ventricle from the aorta. As the
ventricles contract, it opens to allow the oxygenated blood collected in the left
ventricle to flow throughout the body. It closes as the ventricles relax, preventing
blood from returning to the heart.
PHYSIOLOGY
The heart is the muscular organ of the
circulatory system that constantly pumps blood
throughout the body. Approximately the size of
a clenched fist, the heart is composed of
cardiac muscle tissue that is very strong and
able to contract and relax rhythmically
throughout a person's lifetime. The heart has
four separate compartments or chambers. The
upper chamber on each side of the heart, which
is called an atrium, receives and collects the
blood coming to the heart. The atrium then
delivers blood to the powerful lower chamber,
called a ventricle, which pumps blood away
from the heart through powerful, rhythmic
contractions.
The human heart is actually two pumps in one.
The right side receives oxygen-poor blood from
the various regions of the body and delivers it to
the lungs. In the lungs, oxygen is absorbed in
the blood. The left side of the heart receives the
oxygen-rich blood from the lungs and delivers it
to the rest of the body.
Systole. The contraction of the cardiac muscle
tissue in the ventricles is called systole. When
the ventricles contract, they force the blood
from their chambers into the arteries leaving
the heart. The left ventricle empties into the
aorta and the right ventricle into the pulmonary
artery. The increased pressure due to the
contraction of the ventricles is called systolic
pressure.
1. Sinoatrial node (SA
The Sinoatrial Node (often node)
called the SA node or sinus 2. Atrioventricular node
node) serves as the natural (AV node)
pacemaker for the heart.
Nestled in the upper area of the 3. Common AV Bundle
right atrium, it sends the 4. Right & Left Bundle
electrical impulse that triggers Branches
each heartbeat. The impulse
spreads through the atria,
prompting the cardiac muscle
tissue to contract in a
coordinated wave-like manner.
The impulse that originates
from the sinoatrial node strikes
the Atrioventricular node (or AV
node) which is situated in the
lower portion of the right
atrium. The atrioventricular
node in turn sends an impulse
through the nerve network to
the ventricles, initiating the
same wave-like contraction of
the ventricles.
The electrical network serving
the ventricles leaves the
atrioventricular node through
the Right and Left Bundle
Branches. These nerve fibers
send impulses that cause the
cardiac muscle tissue to
contract.
A. Laboratory Result
and significant
HEM AT OLO GY
Significances:
Hematology:
Hgb: still at normal ranges.
Hct: acute massive blood loss
RBC: decreasing due to side effects of the drugs.
WBC: Increasing due to immunocompromised, immune
responses.
Platelet: increasing the fibrin that attract the platelet to
increased
Blood indices:
MCHC: decreased in severe hypochromic anemia.
Coagulation:
Bleeding time: defective in platelet function
INR: prolonged in deficiency of fibrinogen; used to
standardized the prothrombin time and anti- coagulation
therapy.
Serum enzyme levels:
Na+ : decreased; myxedema
K+ : decreased; GI losses, Vitamin D Deficiency
Cl+ : decreased; pneumonia, febrile condition.
Creatinine: decreased; check the status of the kidney
Troponin: negative; if increased the patient may experience
myocardial infarction.
IV. Pathophysiology
& Schematic Diagram
In an MI, an area of the myocardium is permanently destroyed; a condition in which the
blood supply to the heart muscle is partially or completely blocked. The heart muscle
needs a constant supply of oxygen-rich blood. The coronary arteries, which branch off the
aorta just after it leaves the heart, deliver this blood. MI is usually caused by the reduced
blood flow in a coronary artery of an atherosclerotic plaque and subsequent occlusion of
the artery by a thrombus. Coronary artery disease can block blood flow, causing chest
pain. In unstable angina and acute MI are considered to be the same process but different
appoints along a continuum. specifically coronary atherosclerosis (literally “hardening of
the arteries,” which involves fatty deposits in the artery walls and may progress to
narrowing and even blockage of blood flow in the artery., As an atheroma grows, it may
bulge into the artery, narrowing the interior (lumen) of the artery and partially blocking
blood flow. With time, calcium accumulates in the atheroma. As an atheroma blocks more
and more of a coronary artery, An atheroma, even one that is not blocking very much
blood flow, may rupture suddenly. The rupture of an atheroma often triggers the formation
of a blood clot (thrombus), the supply of oxygen-rich blood to the heart muscle
(myocardium) can become inadequate. The blood supply is more likely to be inadequate
during exertion, when the heart muscle requires more blood. An inadequate blood supply
to the heart muscle (from any cause) is called myocardial ischemia. If the heart does not
receive enough blood, it can no longer contract and pump blood normally. Other causes of
MI include vasospasm, (sudden constriction or narrowing) of a coronary artery, decreased
oxygen supply (e.g. from acute blood loss, anemia, or low blood pressure), and increased
demand for oxygen (e.g. rapid heart rate, thyrotoxicosis, or ingestion of cocaine). In each
case, a profound imbalance exists between myocardial oxygen supply and demand. The
area of infarction develops over minutes to hours. As the cells are deprived of oxygen,
ischemia develop, cellular injury occurs,, and the lack of oxygen results in infarction, or
the death of cells. The area of the heart muscle supplied by the blocked artery dies.
Aging
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adventitious breath Ineffective After of nursing assess, document & can be used as a After of nursing
sounds airway intervention report to the guide for intervention the
changes in respiratory physician on activity
clearance the patient abnormal breath prescription patient will clear
rate and rhythm related to will clear the airway patency.
sound and a basis
copious the airway maintain the patency of for patient
tracheobro patency. oxygenation health
nchial therapy management.
Monitor Arterial Blood to provide an
secretions. Gases Analysis oxygen
suction tracheobronchial needed by the
secretion physiologic
established the turning need of the
patient as and body.
“tapping back” , as to indicate the
prescribed by the effectiveness
physician. of
oxygenation
therapy and
changes that
need to
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exchange.
retention of
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lead to
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supply
help to loosen the
secretions.
ASSESSMENT NURSING PLAN OF CARE INTERVENTION RATIONALE EVALUATION
DIAGNOSE
S
Objective: risk for excess fluid After of 8 hours of >AUSCULTATE BREATH > MAY INDICATE After of 8 hours of nursing
>Decreasing urinary volume, nursing SOUNDS FOR PULMONAR intervention the
decreased intervention PRESENCE OF Y EDEMA patient had monitor
output
organ the patient CRACKLES. SECONDARY fluid status and reduce
>abnormal breath TO CARDIAC
perfusion will monitor occurrence of fluid
sounds, crackles DECOMPENS
fluid status excess. the goal is met.
>dyspnea ATION.
and reduce > Measure I&O, noting
occurrence of > DECREASED
decrease in output,
CARDIAC
fluid excess. concentrated
OUTPUT
appearance. Calculate
RESULTS IN
fluid balance.
IMPAIRED
KIDNEY
PERFUSION,
SODIUM/WA
TER
RETENTION,
AND
REDUCED
URINE
OUTPUT.
>assess for edema and > Sudden changes in
weigh daily. weight reflect
alterations in
fluid balance.\
>Sodium enhances
>Provide low-sodium fluid retention
diet/beverages. and should
therefore be
restricted
during active
MI phase
and/or if heart
failure is
present.
ASSESSMENT NURSING PLAN OF INTERVENTION RATIONALE EVALUATION
DIAGNO CARE
SES
Objectives: impaired skin After rendering of assess, document the for guiding data. After rendering of nursing
physical integrity nursing skin patient. to avoid possible care intervention
immobilization related to intervention ask the physician if the that can the patient will not
prolonged bed prolonged the patient patient will trigger to his be able to get a bed
pressure allowed to turn the disease.
bed will not be sore.
patient on side-to to avoid possible
pressure. able to get a side and the time complication
bed sore. interval. on skin.
do the skin care
POTENTIAL PROBLEM
POTENTIAL CONSIDERATIONS following discharge from care setting
(dependent on patient’s age, physical condition/presence of complications,
personal resources, and life responsibilities)