Professional Documents
Culture Documents
Dental Emergencies
Emergencies
EMPA Residency
UTHSCSA
Dental Emergencies
t.1484
Dental Pain is probably one of the least
satisfying aspects of Emergency
Medicine
masticator space
– Associated trismus suggests extension into the
parapharyngeal spaces
• Consider CT neck to characterize size of abscess
• Panorax
• Antibiotics: PCN-VK, Clindamycin, analgesia
• OMS consult
• These infections can progress and obstruct the airway -
Think Ludwigs Angina
Dental Caries
• Extremely Common
• Caused by acidic by products of plaque bacteria
• Continued neglect leads to irreversible damage
to the pulp associated with longer lived pain
• Treatment for irreversible pulpitis is root canal
or extraction
Triage PA’s work fast but
remember lessons learned
in Resusitation
Patient with left mandible pain, 55
years old, and associated SOB
Ludwigs Angina
• Infection of submental, sublingual and
submandibular spaces:
• Rapid upward posterior displacement of tongue
with the potential for complete obstruction of
the airway
• Recussitation Room for emergency airway
management as needed, antibiotics, ENT for
fiberoptic evaluation, CT neck if time allows.
Postextraction Alveolar
Osteitis
• Periosteitis - Occurs within 48 hours of
extraction and responds well to analgesics
• Postextraction Alveolar Osteitis – Dry Socket –
usually occurs after the 2nd or 3rd postoperative
day and causes exquisite pain
– Caused by displacement or disolution of the clot
– Results in exposure of alveolar bone and localized
osteomyelitis
Postextraction Alveolar
Osteitis
Dental radiographs to exclude retained root tip
Thorough irrigation of the socket
Packing with oil of cloves or eugenol gauze results in
almost immediate improvement in comfort
Dental anasthesia may be necessary to pack
Antibiotics for the worst cases, analgesics
24 hour referral to dentist
More common in 3rd molar
Trismus postoperatively is common and should peak
after 24 hours if it worsens after this time it is
worrisome for infection of the TMJ
Periodontal Pahtology
• Periodontitis is generally secondary to gingivitis
• Plaque along the gingival margin causes progressive
inflamation ultimately affecting the periodontal
attachment apparatus. The gingival sulcus also deepens.
• The net result is bone and tooth loss
• Periodontal abscess is common –pcn-vk, analgesics,
possible I&D
Acute Necrotizing Ulcerative
Gingivitis(AKA-Vincent Disease
or Trench Mouth)
• Ulcerative local lesions spread to surrounding tissues
including bone with occasional fatal outcome
• Anaerobic bacteria – Treponema, Selenomonas,
Fusobacterium, Prevotella are uniformly identified
• Predisoposing conditions HIV, previous history of
ANUG- Also, poor, young, poor hygiene, malnourihed,
white, etoh, tobacco
• Clinical triad of pain, puched out interdental papillae
and gingival bleeding
Acute Necrotizing Ulcerative
Gingivitis(AKA-Vincent Disease
or Trench Mouth
• Very difficult to differentiate from
herpes gingivostomatitis
• Treatment with Metronidazole and
chlorhexidine rinses are mainsty
• Surgical debridement may be
necessary
Dental Fractures
• Ellis Classifications: types I-III
• Type I: Involves enamel portion only
– Generally no ED treatment needed.
Ellis Classifications:
types I-III
70% are type II
• Type II: involves the dentim of the tooth
• Dentim is creamy yellow when exposed communication
with the pulp and infection are concern
• Painful stimuli includes hot, cold and even air passing
over tooth when breathing
• Covering the exposed dentim to decrease the chance of
pulpal infection with a dental cement
• Followed by 24 hour referral to dentist to ensure tooth
vitality
Ellis Classifications:
types I-III