Professional Documents
Culture Documents
Lens
The lens is
avascularIt does not have a
vascular supply
Structure
The basic lens consists
of a central nucleus
surrounded by the
cortex contained
within the lens
capsule
Opti
c
Opti
c
Increased nearsightedness,
requiring frequent changes in
your eyeglass prescription
Patients often
describe trying to
look through a
fogged-up window
Pen light
shadow test
Pathology
Cataract classification
Age
1. Cataract congenital ; onset before 2-3 months
of age
2. Cataract juvenile
o
Acquired
Stadium of cataract
1. Insipien; vacuoles and water clefts in the
anterior and posterior cortex
2. Immature; the wedge shapes opacities spread
to adjacent fiber cell along the length of
affected fibers, causing the degree of opacity to
increase and extend toward the visual axis
3. Mature; entire cortex from the capsule to
nucleus becomes white and opaque. If the lens
takes up water, swelling become an
intumescent
4. Hypermature Cataract
Phacoemulsification
During
phacoemulsification,
phaco for short, the
surgeon makes a small
incision, where the
cornea meets the
conjunctiva
Follow-up:
Patients are usually examined
1st, 2nd, 3rd day,
1 week, and
1 month after the surgery date
Conjunctivitis
(Red Eye)
Dr. Delfi, Mked (Oph), Sp. M (K)
Departemen Ilmu Kesehatan Mata
Fakultas Kedokteran Universitas
Sumatera Utara
Tarsal
conjunctiv
a
Fornix
conjunctiv
a
Bulbar
conjunctiv
a
Conjunctivitis is an inflammation or
infection of the transparent membrane
(conjunctiva) that lines the eyeball that
causes the eye to be swollen and makes
small blood vessels in the eye become more
prominent resulting in red eye.
Acute conjunctivitis
Symptoms
The most common symptoms of conjunctivitis
include:
Causes
Causes of red eye include:
Viruses
Bacteria
Allergies
Chemicals
A foreign object
Management
Diabetic
Retinopat
hy
Dr. Delfi, Mked (Oph), Sp. M (K)
Departemen Ilmu Kesehatan Mata
Fakultas Kedokteran Universitas
Sumatera Utara
Foster S.Resnikoff. The impact of vision 2020 on global blindness. Eye 2005; 19:1133-1135
Epidemiology
The prevalence of all types of diabetic
retinopathy in the diabetic population
increases with the duration of diabetes and
patient age.
Pathogenesis
Hyperglycemia over an extended period
Endothelial damage
Capillary occlusion
Symptoms
Proliferative diabetic
retinopathy
Retinal
neovasculariza
tion
Fluorescein
angigraph : dye
leak
Typically sign
neovascularizatio
n
PDR with
Clinically
significant macular
edema (CSME)
After laser
treatment
(laser burn
appears whitish
brown)
Diabetic macular
edema
Rubeosis iridis
Diagnosis
Slit-lamp biomicroscopy
Indirect ophthalmoscopy
Treatment