ERITROPAPULO

SKUAMOSA DERMATOSIS
 ERITROPAPULOSKUAMOSA DERMATOSIS
Golongan penyakit kulit

Effloresensi terutama : eritema, papula, skuama

Penyebab diketahui :
Dermatomikosis superfisialis
Lues II
MH

Penyebab tak diketahui :

Psoriasis vulgaris
Pytiriasis rosea
Dermatitis seboroik
Lichen planus
Pityriasis Rubra Pilaris
PSORIASIS VULGARIS

DEFINISI :
penyakit kulit, kronis dan residif
makula eritematus bulat / lonjong
skuama tebal, transparan/putih keabu-abuan
ETIOLOGI DAN PATOFISIOLOGI
Penyebab pasti tak diketahui
Faktor-faktor :
Pemendekan turn over epidermis
Predisposisi genetik
Faktor pencetus :
- infeksi - obat tertentu
- stres - perubahan iklim
- trauma

EPIDEMIOLOGI
Semua usia, dewasa >>
Pria = wanita
GEJALA KLINIS
Keluhan
- sedikit gatal (panas / burning sensation)
- kosmetik.

Kulit
Makula eritematus, batas jelas, tertutup skuama tebal,
transparan, lepas dibagian tepi, lekat dibagian tengah.
Kaarsvlek phen (+), Austpitz sign (+), Koebner phen (+)
Bentuk makula bisa berubah
anular, gyrata, folikular, gutata, punctata
PREDILEKSI
Tempat mudah kena trauma
( siku, lutut, sakrum, kepala, genitalia)
Kuku :
keruh, pitting, punctate
subungual hiperkeratosis
kuku tangan >>

Mukosa :
geographic tongue

Sendi :
Psoriasis arthropatica
(interphalang dan lumbosacral)
 HISTOPATOLOGI
- Khas, diagnosis pasti
- Hiperkeratosis,
parakeratosis,akantosis,
pemanjangan Rete ridges,
permanjangan papila
dermis, mitosis dari stratum
basalis, mikro abses
Munro
DIAGNOSIS
klinis dan histoPA

DIAGNOSIS BANDING
MH type T
Lues II
Dermatomikosis superfisialis
Dermatitis seboroik
Pytiriasis Rosea
PENYULIT
Eritrodermi

PENATALAKSANAAN
kausal (-)
tujuan : menghilangkan faktor pencetus/lesi
topikal : kortikosteroid, tar, anthralin, vit D,
asidum salisilikum.
sistemik : metotrexate, retinoid, fototerapi
 PSORIASIS PUSTULOSA
Dibagi 2 :
Palmo plantar
Generalisata (von Zumbusch)
Dengan atau tanpa didahului Psoriasis Vulgaris
Klinis :
Febris tingi, KU lemah
Eritema menyebar luas ditumpangi adanya pustule steril.
Pustule bisa bergabung membentuk danau-danau yang
berisi pus.
Laboratorium :
Leukositosis (DL)
PMN >> dan pustule steril tidak didapatkan bakteri (Gram)
Terapi :
MTX, Siklosporin, Asitretin, Kosrtikoseroid oral
 PITYRIASIS ROSEA
DEFINISI
- penyakit kulit, ringan, swasirna
- makulae eritematus, oval, papulae, plak
- skuama disekeliling tepinya

ETIOLOGI DAN PATOFISIOLOGI

- penyebab pasti belum diketahui
- diduga ~ infeksi virus
 Pityriasis rosea
is common self-limited benign
papulosquamous dermatosis associated
with distinctive clinical features.
"Herald patch" followed by a widespread
oval scaly red eruptions.
A viral etiology has been suggested
( human herpesvirus-7 and 6).
 PITYRIASIS ROSEA

EPIDEMIOLOGI
- usia remaja, dewasa muda >>
- pria = wanita

PREDILEKSI
- bagian tubuh tertutup pakaian, leher dagu
- kadang bagian tubuh terbuka Pityriasis Rosea
Inversa
 PITYRIASIS ROSEA
GEJALA KLINIS
- gejala konstitusi
- gatal ringan - sedang / asimtomatik
- herald patch / mother plaque / medalion
- makulae bulat lonjong, tepi meninggi, lekat pd tepi
- sumbu panjang sejajar pelipatan kulit dipunggung
~ gambaran pohon cemara.

HISTOPATOLOGI
- akantosis ringan, parakeratosis fokal
- infiltrat limfosit, perivaskular ringan
 PITYRIASIS ROSEA

DIAGNOSIS
Klinis khas

DIAGNOSIS BANDING
- Psoriasis vulgaris
- Dermatitis seboroik
- Lues II
- Tinea corporis
- MH
 PITYRIASIS ROSEA
PENATALAKSANAAN
- Tx spesifik (-)
- Antihistamin
- Bedak mengandung asidum salicilikum
- Steroid topikal / sistemik bila parah
- Konseling
Pityriasis Rosea, or "PR" for short, is a skin problem of unknown origin. It behaves somewhat like a virus, since most people
get it once in their lives and never again. No one has been able to find an infection agent in most cases studied. Several years
ago, some researchers found a virus-like particle in some skin biopsies.
Pityriasis rosea is:
A. A NUISANCE
B. SELF-LIMITING
C. NON-SCARRING (as long as you don't scratch a lot)
D. NOT SERIOUS
E. NOT DUE TO SOMETHING YOU ATE
F. NOT AIDS
G. NOT VITAMIN DEFICIENCY
H. NOT ANY FORM OF CANCER
I. NOT CONTAGIOUS
There is no need to sterilize your dishes or glasses. You do not have to wash your towels and linens separately from everyone
else, and you do NOT have to isolate yourself from anyone else.
Typical "PR" is characterized by 1-2 centimeter sized oval spots, usually somewhat pink to red, with a little "collar" or scaling. A
"herald patch" that is larger than all the rest comes first in around 50% of cases, and is usually followed by a shower of smaller
ones in several days. They are usually located on your trunk in the folds of the skin. They do NOT usually involve the face,
palms or soles.
Unusual cases of "PR" have fine red bumps, about the size of a mosquito bite, in clusters and patches on the body, face, under
arms, in the groin, and sometimes on your palms. These bumps also develop those little collars of scale over time.
TREATMENT: Typically, a course of "PR" will take 2-3 months to clear if no therapy is used. Erythromycin seems to help in
some cases, may be prescribed and may shorten your course to weeks instead of months. Topical cortisones also help for
symptomatic relief of your itching. Sometimes cortisone injections are given in the buttocks to shorten the course.
Going to the beach and getting into the cool water, and simultaneously getting some sun may also help. Ultraviolet light does
shorten the course. Heat aggravates the itching, so stay cool. Good old- fashioned calamine lotion also seems to help. Apply it
with a paint brush.
2006 Pacific Monograph
 Pityriasis Rosea
My Diary

~Jim Spence ~If you are reading this, more than likely you have just found you that you have Pityriasis
Rosea, or you know someone that does. You've probably been searching web sites, trying to find out
anything you can about it, and you've noticed that there's a lot of information, some of it contradictive,
but no clear cut ideas on what you can actually do about it. Of course, it's impossible to diagnose a
disease on line; however, you can make informed decisions on the information that's available and try
some things yourself to make this disease easier to deal with.
You've probably seen the following information but, just in case you haven't, I'm going to go over some of
the basics. Please know that I'm not a doctor; all I'm doing is compiling the information that I've gotten
from dozens of web sites, a few books, and my own doctor. If you heed any advice that I give you and
harm comes to you shame on you. You should have known better.
The main reason I'm compiling this information is I had Pityriasis Rosea and cured it much quicker
than the norm, in part, I think, because I did my homework. If you're lucky, you can cheat off of me.
Pityriasis Rosea (pit' - e - RYE' - uh - sis RO' - sha) is caused by a virus. What kind of virus is unknown. A
few web sites don't agree with this, but the overwhelming majority (and my personal physician) do, so
that's what I'm going with. The virus enters your body in some unknown manner and manifests itself in a
single spot, usually the size of a nickel, somewhere on your torso, though it can start other places on
your body. In my case, it started on the back of my right thigh.
This spot is called the herald patch. The entire rash that will take place over your body in the next few
months comes from this spot. Sometime between one and 30 days later, you'll notice a rash forming on
the trunk of your body. This rash takes the initial form of a typical bumpy rash, then changes to larger,
wrinkled, pinker spots. The rash usually follows the creases in your skin under your arms, the crease
in your neck, around your navel, on your ribs. The disease, and it's referred to as a disease because it's
viral in nature, is truncated meaning that it usually stays on your torso, down to the shirt sleeve line on
your arms, and to the shorts line on your pants. It reacts differently to different people, of course, but it
usually is a thick covering of your torso, front and back.
Pityriasis Rosea (pit' - e - RYE' - uh - sis RO' - sha) is caused by a virus. What kind of virus is unknown. A
few web sites don't agree with this, but the overwhelming majority (and my personal physician) do, so
that's what I'm going with. The virus enters your body in some unknown manner and manifests itself in a
single spot, usually the size of a nickel, somewhere on your torso, though it can start other places on
your body. In my case, it started on the back of my right thigh.
This spot is called the herald patch. The entire rash that will take place over your body in the next few
months comes from this spot. Sometime between one and 30 days later, you'll notice a rash forming on
the trunk of your body. This rash takes the initial form of a typical bumpy rash, then changes to larger,
wrinkled, pinker spots. The rash usually follows the creases in your skin under your arms, the crease
in your neck, around your navel, on your ribs. The disease, and it's referred to as a disease because it's
viral in nature, is truncated meaning that it usually stays on your torso, down to the shirt sleeve line on
your arms, and to the shorts line on your pants. It reacts differently to different people, of course, but it
usually is a thick covering of your torso, front and back.
The rash can itch, as all rashes do. There is no cure for the disease. All you can do is treat the symptoms
and let it run its course.
The disease is not fatal. The disease is also not communicable no one can catch it from you.

Now, here's the real kicker the disease can last from six weeks to six months.
My doctor prescribed a cream and told me there wasn't much else I could do about it. He told me to check
web sites even he knew there was some good information out there.
At least my doctor had a sense of humor about it. After he let me know what it was, he told me that they
call it Pityriasis Rosea because "we pity the poor son of a bitch that gets it".
So, he sent me home with a cream, and a rash that was beginning to cover over half of my body.
If you read anything about this disease, you'll notice that it predominately stays on your trunk, rarely
going down your arms or legs, or much further than your neck. In my case, the rash spread all the way
down to my wrists, almost getting on the palms of my hands, and down my legs just above my ankles. It
also ran up the back of my neck into my hair and up the front of my neck to my chin. I was covered with it.
I had to have a layer of cream applied over virtually my entire body twice a day.
There are three things that aggravate the rash, besides scratching it sweat, hot water, and soap. Hot
water, in fact, is painful. The first days of your disease, if you take hot showers or baths, you'll notice pain
in areas where you seem to have no rash. This is a good precursor to the rash appearing in these spots
soon.
The worst part is at night. During the day, you can at least give yourself temporary relief with over the
counter creams. At night, you wake up at 2:00 am, itching all over, and can't sleep.
A few of the web sites said some studies were being done, and that they'd had limited success with
ultraviolet light. They said if you have ultraviolet light treatments, make sure they're prescribed by a
physician.
So, you have a disease the stays under your clothes and is killed by ultraviolet light. Since I caught this
disease in early spring, I was still wearing long sleeves and long pants which is why, I believe, the
disease spread as far as it did on my body.
Again, I do not recommend or in any way represent that what I did to rid myself of this disease should be
tried by anyone else. I read the information, made a decision, and tried it and it worked.
DAY 1: I woke up and noticed that I had a sore on the back of my right thigh a small scaly patch about
the size of a penny. It itched a bit, but wasn't overly troublesome. In fact, by the next day I didn't really
think about it.
DAY 4: My butt started to itch a bit. You could see the beginnings of a rash.
DAY 5: The rash had become a bit worse, and had spread around my body to the crease between my
thighs and my torso on the front of my body.
I called my doctor, but he was booked. I agreed to see a nurse practitioner. My best guess was that I'd
gotten this rash from wearing clothing with some kind of mites (since I'd worn a pair of shorts that I
hadn't worn in months a few days before). The nurse agreed with my assessment, gave me a shot of
steroids, a prescription for some cream, and a handful of antihistamines which, I found out, can relieve
itching a bit.
DAY 8: The rash had spread to my shoulder blades, and was beginning to make its way under my arms. It
also was appearing on my chest, and had become much thicker on my butt and above my thighs.

DAY 9: This rash obviously wasn't caused by mites so I saw my physician at 9:00 in the morning. I told
him about the rash, what the nurse had thought it was, and took off my shirt to show him how far it had
spread on my body. His first question was "did you have one spot that you noticed itched before all the
others?" The herald spot. I showed him, he looked at it, and told me about Pityriasis Rosea.
DAY 11: I'd spent the last two days getting covered from head to toe with a particular cream that was a bit
stronger than the normal prescription. The rash continued to cover my body and was now making its way
up the back of my neck, through my arm pits, and down my arms. Also, I started to get a low grade fever
that would last for the next five days.
DAY 13: I finally discovered the correlation between hot water and the disease. I started taking the coldest
showers possible, using soap only on my face and my hair.
DAY 16: My back was completely covered. My chest was showing signs of complete coverage within a
few days, my stomach was starting to get the small bumps, and the rash had made its way to the bend in
both elbows. The worst itching came from my navel; the rash had it covered.
For the past week, I hadn't slept more than three hours at a time. I woke up, itching badly. I would apply
some over the counter medicine to various spots that itched, but I'd be back up in three hours or less
doing the same thing again.
I was still taking cold showers with no soap, but this time I broke down and used soap on my entire body.
The rash was redder than ever before, and itched worse than it had previously.
DAY 17 - 18: The rash continued to grow. I was covered from head to toe twice a day with prescription itch
cream. During the day I would cover particularly itchy spots with over the counter medicine. I was still
waking up two or three times a night, applying over the counter medicine to spots that itched so badly
they woke me up.
DAY 19: The rash still had my entire back covered, by now in large, pink wrinkled masses. The rash was
completely under both arms, all the way down both arms to my wrists; in fact, my wrists were the worst
spot on both arms. The rash had also made its way down my legs, well past my knees, and looked a lot
like my wrists. A huge rash was growing up the front of my neck, and had made its all the way to my chin.
It had grown up the back of my neck, well into my hair. Luckily, the rash didn't find its way to my
"sensitive areas", but I was afraid that it would soon. It was itching tremendously, and showed no signs
of slowing down. By this time I'd gone 10 days without much sleep and nothing but cold showers.

I felt like the elephant man. I was embarrassed to go out in public because the rash was becoming quite
visible.
This was when I made up my mind to try something.
I went to a tanning salon and bought a month's pass. I spent 15 minutes on a tanning bed that evening
(completely naked, save for my well, my manhood).
DAY 20: The rash looked no better, but no worse. It was a warm sunny day, so I mowed the grass with no
shirt on. The sweat, and the grass trimmings and pollen that stuck to the sweat, made me itch worse than
I had so far. A cold shower helped that.
DAY 21: I spent 20 minutes on a tanning bed (as naked as the first day). The rash was beginning to show
signs of stopping its forward advance. One spot, the huge rash that was growing up my neck to my chin,
had in fact begun to dwindle away.
DAY 23: I spent another 20 minutes on a tanning bed, this time wearing just my briefs. The heaviest part
of the rash on my back was diminishing. The rash on my neck was just a small mark. The rash itched less
than it had in two weeks. The rash on my butt, the original rash, was now nothing more than small dark
bumps, and didn't itch much at all. Same as the rash that was on the front crease of my thighs. For the
first night, I went to bed without using any cream. I still got up once during the night to apply some over
the counter medicine, but after falling back to sleep, I slept for six hours the longest stretch of sleep I'd
had in two weeks.
DAY 24: I didn't use cream again this morning. The rash under my arms now looked like the rash on my
butt the previous day; small, dark spots that don't particularly itch. The rash on my lower thighs, below
my knees, was thinning out. The largest spots, on my back over my shoulder blades, were now just
blotchy red spots, and itched less than usual. The rash that was making its way up the front of my neck
just five days before was almost completely gone. The rash was almost gone from my hair, but was still
hanging on; I reasoned that was because the tanning bed couldn't get through my hair completely.
I took a warm shower, and used soap on my entire body, for the first time in 12 days.

DAY 25: Three weeks ago today I noticed the first rash on my butt. Since all of the information I'd gotten
said the rash would last from six weeks to six months, I should be, at best, halfway through the disease;
meaning that the disease should, as of today, be at just about it's worst point. It was obvious that the
rash, and the disease, was subsiding much faster than it should have been.
I took another warm shower with soap, and didn't have to apply any cream to what was left of the rash. It
itched a bit, but nowhere near as badly as it did just a week before. Eventually you get used to minor
itching and pay it no mind.
I spent another 20 minutes on the tanning bed.
DAY 26: The rash on my neck is completely gone. The rash in my hair is virtually gone, save for a few
small spots. The rash on my butt and the front of my thighs is nothing more than small, dark spots that
don't itch. The rash down my thighs is almost gone, save for a few dark red spots, and they don't itch. The
rash on my shoulder blades, the worst areas affected, continued to thin out, becoming darker and more
pronounced.
Another 20 minutes on the tanning bad, and every spot on my body continued to heal; the small amount
of rash in my hair was still holding on, but sure wasn't growing.
DAY 27: I was winning the battle. I hadn't applied prescription cream in four days, except on the
occasional spots that would itch, and those were rare
DAY 30: I went an extra day between tanning appointments. I was by now quite dark, and the rash was on
the run. Another 20 minutes on the tanning bed. The rash in my hair was almost gone.
DAY 32: Four weeks ago today the first rash started. Now it was totally gone. In four weeks. Six tanning
bed sessions in two weeks had killed it. Now, I'm sure there are some in the medical profession that
would disagree with me, saying that perhaps I had had a mild case of the disease. To that I say horse
hockey. I was eaten up with it, it was growing by leaps and bounds and, from the second trip to the
tanning salon, I noticed it diminishing.
I killed it. With a half dozen sun tans.
Again, I make no claims or representations that what I did in any way affected this disease. But, in my
heart, I know it did.
I rid myself of a disease that can last up to six months in four weeks and got one hell of a tan in the
process.

Jim Spence
 DERMATITIS SEBOROIK
DEFINISI
penyakit kulit, radang superfisialis, kronis
predileksi area seboroik
remisi dan eksaserbasi
ETIOLOGI
Penyebab pasti belum diketahui
Dugaan : - Pityrosporum ovale >>
- stres
- kasus parah HIV (AIDS),
penyakit neurologis
Status seboroika yang diturunkan
 DERMATITIS SEBOROIK

EPIDEMIOLOGI
Usia bayi dan orang dewasa
pria >>

PREDILEKSI
area seboroik - kelenjar sebasea >> :
kepala, wajah, badan atas, pelipatan
 DERMATITIS SEBOROIK

HISTOPATOLOGI
Akantosis, spongiosis ringan,
Infiltrat sel-sel radang, perivaskular
DIAGNOSIS
Klinis khas
DIAGNOSIS BANDING
Psoriasis vulgaris
Pityriasis rosea
Tinea corporis
 DERMATITIS SEBOROIK

PENYULIT
Rambut rontok
Infeksi sekunder
Eritroderma

PENATALAKSANAAN
Diit rendah lemak
Faktor pencetus dihindari
Topikal
Kepala : shampo selenium sulfide 1,8%, Zn pirithion,
ketokonazole scalp solution 1%/2%, losio steroid
Lokasi lain : krim kortikosteroid, Imidasol
Cradle Cap pada scalp bayi
 LICHEN PLANUS (LP)

DEFINISI :
- inflamasi kulit, folikel rambut, membrana mukosa,
gatal

ETIOLOGI DAN PATOFISIOLOGI

penyebab belum diketahui dengan pasti
diduga : - kelainan imunologi
- ~ infeksi bakteri, virus, jamur
- paparan obat, bahan kimia tertentu
 LICHEN PLANUS (LP)

GEJALA KLINIS
Gatal
Kulit : papulae ( 1-4 mm) permukaan rata, kering,
berkilat, poligonal, keunguan, sedikit skuama
Wicham's Striae (+)
penyembuhan hiperpigmentasi
ekstremitas bawah >> Koebner phen (+), bilateral
simetris
Kuku : pterygeum, onikolisis proksimal & distal,
lekukan longitudinal
 LICHEN PLANUS (LP)

GEJALA KLINIS
Mukosa : oral >> bentuk :
- Ulseratif >>
- Reticulate : pipi bagian dalam >>
- Atropik
Genitalia :
glans penis, labia, anus papulae poligonal
- Vulvovaginal erosif / ulseratif
- Varian : Linear, Anular, Folikular,
Hipertropik, Ulseratif, Bollous
 LICHEN PLANUS (LP)

DIAGNOSIS
klinis

DIAGNOSIS BANDING
- Psoriasis vulgaris
- Candidiasis
- Lues II
- Pityriasis Rosea
 LICHEN PLANUS (LP)

PENATALAKSANAAN
Lesi terbatas :
topikal steroid superpoten / injeksi intralesi
Lesi tersebar :
sistemik steroid, PUVA, retinoid
Lesi oral :
steroid in orabase,
injeksi intralesi
 LICHEN PLANUS (LP)

HISTOPATOLOGI
awal dermatitis pada taut dermoepidermal
selanjutnya : hiperplasia epidermis, pola saw tooth
orthokeratosis, hipergranulosis
lapisan basal : keratinosit nekrotik(+)
dermis : civatte bodies (+)
 Ada dua jenis manusia di dunia ini,
seorang realis dan pemimpi.
Mereka yang realis tahu kemana akan pergi.
Mereka yang pemimpi telah tiba di sana

Robert Orben
 PITYRIASIS RUBRA PILARIS (PRP)

DEFINISI
Kelainan menahun dengan :
- Plak eritematus
- Berskuama
- Papul keratotik folikuler

ETIOLOGI
- HEREDITER
- Autosomal dominan
- Permulaan pada masa anak
- DIDAPAT
- Semua umur
- Riwayat keluarga (-)
- Diduga defisiensi vitamin A
 PITYRIASIS RUBRA PILARIS (PRP)

KLASIFIKASI

Herediter
- Meluas bertahap dan perlahan
- Menetap

Didapat
- Meluas dengan cepat
- Remisi
 PITYRIASIS RUBRA PILARIS (PRP)

GEJALA KLINIS
Eritema dan skuama pada permulaan di muka
dan kulit kepala yang bisa meluas ke seluruh
permukaan kulit, dilanjutkan eritema dan
penebalan di telapak tangan dan kaki.
Kelainan kulit batas tegas dengan pulau-pulau
kulit normal.
Kuku menunjukkan penebalan.
Kelainan sistemik (-).
 PITYRIASIS RUBRA PILARIS (PRP)

HISTOPATOLOGIS
Hiperkeratosis
Parakeratosis
Akantosis
Sebukan sel radang menahun pada dermis
atas.
 PITYRIASIS RUBRA PILARIS (PRP)

TERAPI

Vit A 200.00 unit per hari.

Asam retinoat 0,05%
Kortkosteroid topikal + Asidum salisilikum
3 20%
 PITYRIASIS RUBRA PILARIS (PRP)

PROGNOSIS

Herediter : buruk
Didapat : baik
 ERITRODERMA

DEFINISI
Kelainan kulit ditandai adanya eritema di
hampir seluruh tubuh, biasanya disertai
skuama.

SINONIM
Dermatitis Eksfoliativa
 ERITRODERMA

PATOFISIOLOGI
Tergantung faktor penyebab.
Pada Eritroderma terjadi peningkatan
epidermal turn over, sehingga terbentuk
skuama berlebihan.
Skuama terutama mengandung protein,
asam nukleat dan asam amino bebas.
 ERITRODERMA

GEJALA KLINIS
Timbul dalam waktu singkat
Cepat meluas
Demam, menggigil,malaise
Seluruh kulit tampak eritematus, mengkilap,
mengelupas, teraba panas, gatal, kasar dan
menebal.
 ERITRODERMA
PATOFISIOLOGI
Eritema : pelebaran pembuluh darah
aliran darah ke kulit meningkat
kehilangan panas bertambah :
Penderita kedinginan / menggigil
Hipotermi (akibat peningkatan perfusi
kulit)
Dehidrasi (akibat penguapan cairan yang
meningkat)
Pengaturan suhu terganggu.

Kehilangan skuama dapat menyebabkan kehilangan

protein (hipoproteinemia) dan oedema.
 ERITRODERMA

GEJALA KLINIS
Alergi obat secara sistemik
- Anamnesis : riwayat minum obat/jamu.
- Alergi timbul secara akut (kurang lebih 10 hari).
- Berupa eritema universal tanpa skuama,
setelah sembuh baru timbul eritema.

Perluasan penyakit kulit

Psoriasis :
- Karena penyakitnya sendiri atau pengobtan yang
terlalu kuat.
- Eritema yang tidak merata.
(Eritema meninggi pada tempat predileksi psoriasis)
 ERITRODERMA
Dermatitis Seboroika pada bayi (Leiners disease)
- Dermatitis seboroika yang meluas
- Etiologi belum diketahui dengan jelas
- Usia : >>> 4 20 minggu.
- KU penderita baik.
- Eritema universal disertai skuama kasar.

Penyakit sistemik termasuk keganasan

- Digolongkan dalam CTCL (Cutaneous T-Cell Lymphoma)
- >>> pada manula.
- Eritema universal meah membara + skuama dan sangat
gatal.
- Pada 1/3 penderita : splenomegali, limfadenopati
superfisial, alopesia, hiperpigmentasi, hiperkeratotik
palmoplantar, kuku distrofia.
- Laboratorium : Limfoma atipik disebut sel sezary
 ERITRODERMA

DIAGNOSIS
Klinis ditemukan keradangan kulit yang eritematus
disertai deskuamasi hingga 90% luas permukaan
tubuh.

DIAGNOSIS BANDING
Dermatitis, Psoriasis vulgaris, Drug
Eruption,Limfoma/Leukemia,Pemfigus, Pityriasis Rubra
Pilaris,Likhen Planus, Dermatofitosis, Skabies
 ERITRODERMA

PENYULIT
Hipotermi
Dekompensasi kordis
Kegagalan sirkulasi perifer
Tromboflebitis
Infeksi sekunder pada kulit dan paru
 ERITRODERMA

PENATALAKSANAAN
Perbaiki cairan tubuh
Eliminasi faktor-faktor pencetus
Kortikosteroid sistemik tappering off
Antibiotika untuk mencegaf infeksi sekunder
Antihistamin/antipruritus
Topikal : emolient (oleum coccos, hidrokortison 1%)
Diit tinggi protein
Seni yang dilakukan
oleh DERMATOLOG adalah
memberi suatu penyakit dengan
nama latin yang panjang ..
dan kemudian memberinya
steroid topikal.